Which central nervous system structure is associated with anger and aggression?
Amygdala.
Parietal lobe.
Cerebellum.
Basal ganglia.
The Correct Answer is A
Choice A reason: The amygdala regulates emotional responses like anger and aggression, with excessive norepinephrine and glutamate signaling driving hyperarousal. Its connections to the prefrontal cortex modulate impulsive behaviors, making it the primary structure for aggressive responses in emotional dysregulation.
Choice B reason: The parietal lobe processes sensory and spatial information, not emotions like anger. Aggression involves amygdala-prefrontal cortex interactions, not parietal functions, making this structure irrelevant to the neural basis of anger and aggressive behavior.
Choice C reason: The cerebellum coordinates motor activity and balance, not emotional regulation. Anger and aggression stem from amygdala-driven hyperarousal, not cerebellar function, which lacks direct involvement in emotional processing or behavioral impulsivity.
Choice D reason: The basal ganglia modulate movement and reward, not directly anger or aggression. While dopamine dysregulation may contribute, the amygdala’s role in emotional arousal is primary, making the basal ganglia secondary in this context.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A reason: Olanzapine does not primarily increase serotonin or norepinephrine. It blocks dopamine D2 and serotonin 5-HT2 receptors, reducing hallucinations driven by mesolimbic dopamine excess in schizophrenia. Increasing monoamines would exacerbate psychosis, not alleviate it, making this mechanism incorrect.
Choice B reason: Olanzapine, an atypical antipsychotic, blocks dopamine D2 receptors in the mesolimbic pathway, reducing excessive dopamine signaling that causes hallucinations in schizophrenia. It also modulates serotonin 5-HT2 receptors, balancing limbic activity, making this the primary mechanism for controlling psychotic symptoms.
Choice C reason: Decreasing neurotransmitter breakdown enzymes is the mechanism of MAOIs, not olanzapine. Olanzapine’s dopamine and serotonin receptor blockade reduces psychotic symptoms, not enzyme activity, making this choice irrelevant to its antipsychotic action in schizophrenia.
Choice D reason: Olanzapine does not normalize serotonin, norepinephrine, and dopamine levels but blocks their receptors, particularly dopamine D2, to reduce hallucinations. Normalizing levels is not its mechanism, as schizophrenia involves dopamine hyperactivity, not deficiency, making this choice inaccurate.
Correct Answer is B
Explanation
Choice A reason: Questioning death’s reason reflects normal grief, involving amygdala-driven emotional processing and cortisol surges. It does not indicate complicated bereavement, which involves prolonged, impairing despair, disrupting serotonin-mediated emotional recovery after a year.
Choice B reason: Stating life has no meaning a year after loss suggests complicated bereavement, with persistent serotonin deficits and amygdala-driven despair. This indicates unresolved grief, impairing prefrontal cortex-mediated coping and emotional integration, characteristic of prolonged, dysfunctional mourning.
Choice C reason: Questioning God’s role is a normal grief response, reflecting amygdala-driven existential distress. It does not indicate complicated bereavement, which requires persistent functional impairment and serotonin dysregulation beyond the expected grief timeline of one year.
Choice D reason: Self-focused questioning is part of normal grief, driven by amygdala-anxiety and cortisol. It does not signify complicated bereavement, which involves prolonged, debilitating despair and serotonin deficits, impairing recovery and functioning beyond a year.
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