Which patient meets the criteria for hospice services?
A 74-year-old newly diagnosed with chronic obstructive pulmonary disease (COPD) and life expectancy of 2 years.
A 16-year-old with type 1 diabetes, multiple infections, and substance abuse.
A 36-year-old diagnosed with multiple sclerosis complicated by major depressive disorder and pain associated with muscle spasms.
A 54-year-old diagnosed with glioblastoma and life expectancy of 8 to 10 weeks.
The Correct Answer is D
Choice A reason: Hospice requires a prognosis of 6 months or less. COPD with a 2-year life expectancy does not meet this, as the chronic disease allows longer-term management, not immediate palliative care for end-stage illness.
Choice B reason: Type 1 diabetes and substance abuse are not terminal conditions meeting hospice criteria (6 months or less prognosis). These require chronic management, not end-of-life care, as they lack imminent mortality despite complications.
Choice C reason: Multiple sclerosis with depression and pain is chronic, not terminal within 6 months, excluding hospice eligibility. Symptom management, not end-of-life care, is appropriate, as the prognosis does not indicate imminent death.
Choice D reason: Glioblastoma with an 8-10 week prognosis meets hospice criteria (6 months or less). Rapid tumor progression causes neurological decline, requiring palliative care to manage symptoms and support end-of-life needs, aligning with hospice goals.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason: Citalopram, an SSRI, increases serotonin levels, requiring 4-6 weeks to upregulate serotonin receptors and modulate prefrontal-amygdala circuits for mood improvement. Explaining this delay addresses patient expectations, reducing frustration and enhancing adherence by clarifying the neurochemical timeline of antidepressant action.
Choice B reason: Increasing the dose after 4 days is premature, as SSRIs like citalopram require weeks to alter serotonin signaling and neural plasticity. Premature dose escalation risks side effects like serotonin syndrome without addressing the expected therapeutic lag, making it an inappropriate intervention.
Choice C reason: Assessing for improvement after 4 days is unlikely to yield significant findings, as citalopram’s serotonin modulation takes weeks to impact mood via prefrontal cortex changes. This approach may reinforce the patient’s frustration without addressing the neurochemical basis of the delayed response.
Choice D reason: Reassuring effectiveness without explaining the delay is misleading. Citalopram’s action on serotonin pathways requires time for receptor upregulation and neural circuit adaptation. This vague reassurance does not educate the patient on the neurochemical timeline, potentially reducing trust and adherence.
Correct Answer is B
Explanation
Choice A reason: Absence of symptoms does not indicate the prodromal phase of schizophrenia, which involves subtle cognitive and functional declines driven by early dopamine dysregulation in the prefrontal cortex. This woman shows no signs of prodromal neural changes, making this choice incorrect.
Choice B reason: The prodromal phase of schizophrenia includes decreased concentration and functioning, reflecting early dopamine and glutamate dysregulation in the prefrontal cortex and hippocampus. These subtle neurocognitive changes precede overt psychosis, accurately describing the 20-year-old’s symptoms as prodromal.
Choice C reason: Exacerbation after stopping antipsychotics indicates active schizophrenia, not the prodromal phase. Dopamine receptor hypersensitivity from medication withdrawal causes symptom relapse, not the subtle, pre-psychotic cognitive decline characteristic of the prodromal period, making this incorrect.
Choice D reason: Bizarre and disruptive behavior indicates active schizophrenia, not the prodromal phase. Overt psychosis reflects advanced mesolimbic dopamine hyperactivity, unlike the prodromal period’s subtle cognitive and functional impairments driven by early prefrontal and hippocampal changes.
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