A client is viewing X-rays of a healing bone fracture and asks the nurse if the bone will ever be ‘normal’ again. Based on the nurse’s knowledge of bone healing, what is the best response?
The bone will be more ‘normal’ when there is inflammation and hematoma surrounding the fracture site
The bone will be more ‘normal’ when callus formation occurs
The bone will be more like ‘normal’ when bone-forming cells absorb the callus into woven bone
The bone will be more normal when there is a proliferation of osteoclasts at the site
The Correct Answer is C
Choice A reason: Inflammation and hematoma formation occur immediately after a fracture, initiating healing by recruiting immune cells and growth factors. However, this stage does not restore ‘normal’ bone structure, as it involves soft tissue response, not bone remodeling. This statement is inaccurate, as the bone remains structurally abnormal during this early phase.
Choice B reason: Callus formation, occurring 2-6 weeks post-fracture, involves soft and hard callus bridging the fracture. While critical, it represents a temporary, weaker structure, not ‘normal’ bone. Osteoblasts form a cartilaginous matrix, but full strength and normal bone architecture require further remodeling, making this statement less accurate.
Choice C reason: Woven bone formation, where osteoblasts convert callus into disorganized woven bone, marks progression toward normal bone structure. This bone is later remodeled by osteoclasts and osteoblasts into lamellar bone, restoring strength and architecture. This statement is accurate, as woven bone formation approaches ‘normal’ bone structure during healing.
Choice D reason: Osteoclast proliferation resorbs bone during remodeling but does not directly restore ‘normal’ bone. Excessive osteoclast activity could weaken the bone. Osteoblasts, not osteoclasts, drive the formation of woven and lamellar bone, making this statement inaccurate, as osteoclasts support remodeling, not normalization, of bone structure.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason: Kidney disease does not directly cause constipation. While it may cause fluid imbalances or medication side effects, it is not a primary factor in assessing constipation. Bowel function is more influenced by diet, activity, and medications, making kidney disease irrelevant as a routine assessment tool for this condition.
Choice B reason: Dietary intake, particularly low fiber or inadequate hydration, is a primary cause of constipation. Assessing fiber, fluid, and food types helps identify contributing factors, as low-fiber diets slow intestinal transit. This tool is essential in evaluating constipation, as diet directly impacts stool consistency and frequency.
Choice C reason: Routine physical activity affects bowel motility, as sedentary lifestyles slow peristalsis, contributing to constipation. Assessing activity levels identifies whether lack of movement is a factor. This tool is critical, as exercise promotes intestinal transit, making it a key component in constipation assessment.
Choice D reason: Medication history is vital, as drugs like opioids, anticholinergics, or calcium supplements can cause constipation by slowing gut motility or altering fluid balance. Assessing medications identifies potential causes, making this tool essential for a comprehensive constipation evaluation to guide appropriate interventions.
Correct Answer is B
Explanation
Choice A reason: A blood sugar of 50 mg/dL indicates hypoglycemia, not normal glucose levels (70-110 mg/dL). Symptoms like sweating and clamminess confirm this. Drinking water does not address hypoglycemia, as it lacks glucose to raise blood sugar, making this intervention inappropriate and potentially harmful.
Choice B reason: Hypoglycemia (50 mg/dL) with symptoms like sweating requires rapid glucose correction. Fruit juice with added sugar provides fast-acting carbohydrates (15-20g), raising blood sugar within minutes by stimulating glycogenolysis and glucose absorption. This is the most appropriate intervention to reverse hypoglycemia safely and effectively.
Choice C reason: Administering insulin during hypoglycemia (50 mg/dL) would further lower blood sugar, worsening symptoms and risking seizures or coma. Insulin drives glucose into cells, exacerbating the glucose deficit. This intervention is contraindicated and dangerous in the context of low blood sugar and neuroglycopenic symptoms.
Choice D reason: While consulting a healthcare provider may be needed for recurrent hypoglycemia, the immediate priority is correcting low blood sugar (50 mg/dL) with fast-acting carbohydrates. Delaying treatment by calling first risks prolonged hypoglycemia, potentially causing neurological damage, making this less appropriate than direct intervention.
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