A client with diabetes mellitus develops an ulcer on the bottom of the foot. Which pathology is the primary contributing factor to the ability of the ulcer to heal?
Sensory neuropathy.
Hyperlipidemia.
Ketoacidosis.
Microvascular changes.
The Correct Answer is D
A) Sensory neuropathy:
Sensory neuropathy, while common in diabetes mellitus, primarily affects sensation in the extremities and can lead to reduced pain perception and protective sensation. While sensory neuropathy can contribute to the development of foot ulcers by reducing the ability to detect trauma or pressure, it is not the primary factor influencing the ability of the ulcer to heal.
B) Hyperlipidemia:
Hyperlipidemia, characterized by elevated levels of lipids (cholesterol and triglycerides) in the blood, is a common comorbidity in diabetes mellitus. It can contribute to the development of atherosclerosis and macrovascular complications such as coronary artery disease and peripheral arterial disease. While macrovascular disease can impair wound healing by reducing blood flow to the affected area, it is not the primary contributing factor to the ability of the ulcer to heal.
C) Ketoacidosis:
Ketoacidosis, a serious complication of uncontrolled diabetes mellitus, results from the accumulation of ketones in the blood, leading to metabolic acidosis. While ketoacidosis can have systemic effects and impair overall health, it is not directly related to the ability of a foot ulcer to heal.
D) Microvascular changes:
Correct. Microvascular changes, such as thickening of the capillary basement membrane and endothelial dysfunction, are hallmark features of diabetes mellitus. These changes lead to impaired microcirculation, reduced oxygen delivery, and compromised nutrient supply to tissues, including the skin and soft tissues of the foot. Poor microvascular perfusion contributes to delayed wound healing and an increased risk of infection in individuals with diabetes mellitus. Addressing microvascular changes is essential for promoting wound healing in diabetic foot ulcers, making it the primary contributing factor to the ability of the ulcer to heal.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
DKA is a serious complication of diabetes mellitus characterized by hyperglycemia, ketosis, and metabolic acidosis. The laboratory results consistent with DKA include:
Elevated blood glucose level: A blood glucose level of 525 mg/dL (28 mmol/L) is significantly elevated and consistent with DKA.
Low arterial blood pH: A decreased arterial blood pH indicates acidosis, which is characteristic of DKA. Normal arterial blood pH ranges from 7.35 to 7.45.
Low bicarbonate (HCO3-) level: A low bicarbonate level indicates metabolic acidosis, which is also characteristic of DKA. Normal bicarbonate levels range from 21 to 28 mEq/L (21 to 28 mmol/L).
Among the options provided:
A) Arterial blood pH 7.5 and bicarbonate level 32 mEq/L (32 mmol/L):
This pH and bicarbonate level are indicative of alkalosis, which is not consistent with DKA.
B) Arterial blood pH 7.42 and bicarbonate level 18 mEq/L (18 mmol/L):
This pH is within the normal range, and the bicarbonate level is slightly decreased but not indicative of metabolic acidosis consistent with DKA.
C) Arterial blood pH 7.25 and bicarbonate level 10 mEq/L (10 mmol/L):
Correct. This pH is decreased, indicating acidosis, and the bicarbonate level is significantly below the normal range, consistent with metabolic acidosis characteristic of DKA.
D) Arterial blood pH 7.38 and bicarbonate level 29 mEq/L (29 mmol/L):
While the pH is within the normal range, the bicarbonate level is elevated, which is not consistent with metabolic acidosis seen in DKA.
Correct Answer is A
Explanation
The client's symptoms, along with the elevated serum amylase and lipase levels, suggest the development of acute pancreatitis as a postoperative complication. Here's a detailed explanation for why option A is the correct choice:
A) Acute pancreatitis:
Correct. Acute pancreatitis is characterized by inflammation of the pancreas, which can be triggered by various factors, including gallstones, alcohol consumption, and certain medications. In this case, the client's recent cholecystectomy for cholelithiasis (gallstones) may have led to the development of acute pancreatitis. The persistent upper abdominal pain radiating to the back, along with vomiting and fever, are classic symptoms of acute pancreatitis. Elevated serum amylase and lipase levels are common laboratory findings in acute pancreatitis due to pancreatic cell injury and leakage of these enzymes into the bloodstream.
B) Surgical site infection:
While surgical site infections are potential complications of cholecystectomy, the client's symptoms, including upper abdominal pain, vomiting, and fever, are more indicative of a systemic inflammatory process rather than localized infection at the surgical site.
C) Hepatorenal failure:
Hepatorenal failure, also known as hepatorenal syndrome, refers to kidney dysfunction that occurs as a complication of advanced liver disease. The client's symptoms and laboratory findings are not consistent with hepatorenal failure, as there are no signs of significant liver dysfunction or advanced liver disease.
D) Biliary duct obstruction:
While biliary duct obstruction can lead to symptoms similar to those of acute pancreatitis, such as upper abdominal pain and vomiting, the presence of elevated serum amylase and lipase levels strongly suggests pancreatic involvement rather than isolated biliary duct obstruction.
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