A nurse is providing diabetic teaching for a family of a client newly diagnosed with diabetes. The nurse determines that the family understands the reason for having glucagon on hand for emergency home use if the family indicates that the purpose of the medication is to treat:
Hyperglycemia from insufficient insulin.
Hypoglycemia from insulin overdose.
Diabetic ketoacidosis.
Hyperglycemia occurring on 'sick days.
The Correct Answer is B
Choice A rationale: Hyperglycemia results from a lack of insulin, leading to an inability of cells to uptake glucose. Glucagon functions as a hormone that stimulates the liver to convert stored glycogen into glucose, thereby raising blood glucose levels. Administering glucagon in an already hyperglycemic state would exacerbate the condition, potentially leading to a dangerous spike in blood sugar. It's used for low, not high, blood sugar.
Choice B rationale: Hypoglycemia, a dangerously low blood glucose level (typically below 70 mg/dL), can be caused by an overdose of insulin, which drives too much glucose into cells. Glucagon is a hormone that counteracts insulin's effects by stimulating glycogenolysis in the liver, releasing stored glucose into the bloodstream. This rapid increase in circulating glucose effectively treats the acute hypoglycemic episode, making it an essential emergency treatment.
Choice C rationale: Diabetic ketoacidosis (DKA) is a severe metabolic complication of diabetes characterized by hyperglycemia, ketosis, and metabolic acidosis. It is caused by an absolute or relative lack of insulin. Administering glucagon would increase blood glucose levels even further, worsening the hyperglycemia, which is a primary component of DKA. Insulin and fluid replacement are the mainstays of treatment for DKA.
Choice D rationale: Hyperglycemia during 'sick days' is a common phenomenon in people with diabetes due to increased stress hormones like cortisol and epinephrine, which raise blood glucose. Administering glucagon would further elevate blood sugar, intensifying the existing hyperglycemia. Therefore, glucagon is contraindicated in this scenario, as it would worsen the metabolic state rather than improving it.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Choice A rationale: Diabetic ketoacidosis (DKA) is a severe complication of diabetes characterized by hyperglycemia (glucose > 250 mg/dL), ketonemia, and metabolic acidosis. It typically occurs in type 1 diabetes when there is an absolute insulin deficiency, leading to increased lipolysis and ketone body production. DKA is not associated with low blood glucose levels.
Choice B rationale: Diabetic neuropathy is a long-term complication of diabetes resulting from sustained hyperglycemia, which damages nerves throughout the body. Symptoms can include pain, numbness, tingling, or muscle weakness. This condition is a chronic effect of poor glucose control and is not an acute state caused by a sudden drop in glucose levels.
Choice C rationale: Hyperosmolar hyperglycemic nonketotic syndrome (HHNS) is a serious complication, predominantly in type 2 diabetes, marked by extremely high blood glucose levels (typically > 600 mg/dL), severe dehydration, and hyperosmolarity without significant ketosis. This condition is the opposite of what the client is experiencing, as it involves very high, not low, glucose levels.
Choice D rationale: Hypoglycemia is a condition characterized by abnormally low blood glucose levels, specifically below 70 mg/dL. This occurs when there is an imbalance between insulin action and glucose intake, such as too much insulin, skipped meals, or intense exercise. The brain relies on glucose for energy, and its function is compromised in hypoglycemia, leading to symptoms like confusion, shakiness, and sweating.
Correct Answer is ["A","C","E"]
Explanation
Choice A rationale: Lipodystrophy is a complication of insulin therapy where changes in subcutaneous fat tissue occur at injection sites. Repeated injections in the same area can lead to either lipohypertrophy (a lump of fat) or lipoatrophy (a depression in the skin), which can impair insulin absorption. Rotating injection sites helps prevent this condition.
Choice B rationale: The dawn phenomenon is a physiological event, not a complication of insulin therapy. It is a morning hyperglycemia caused by the normal nocturnal release of counter-regulatory hormones like growth hormone and cortisol, which increase liver glucose production and cause blood sugar levels to rise. This is an endogenous process.
Choice C rationale: Hypoglycemia is a common and serious complication of insulin therapy. It occurs when insulin levels are too high relative to the available glucose, causing blood sugar to drop below 70 mg/dL. This can result from a missed meal, excessive exercise, or an incorrect insulin dose, leading to neuroglycopenic symptoms.
Choice D rationale: The Somogyi phenomenon, or rebound hyperglycemia, is a physiological response to insulin-induced hypoglycemia. An excessive insulin dose causes blood glucose to drop during the night, triggering the release of counter-regulatory hormones that cause rebound hyperglycemia in the morning. This is not a direct complication of the insulin itself but of its administration.
Choice E rationale: Insulin resistance is a complication where the body's cells become less responsive to the effects of insulin, requiring higher and higher doses to achieve the desired glycemic control. This can be caused by various factors, including obesity, inactivity, and the development of antibodies to insulin, impairing its ability to facilitate glucose uptake.
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