A nursing student who is preparing to care for a postoperative patient with deep vein thrombosis asks the nurse why the patient must take heparin rather than warfarin. Which response by the nurse is correct?
The onset of warfarin is delayed.
Warfarin prevents platelet aggregation.
Heparin has fewer adverse effects.
Heparin has a longer half-life.
The Correct Answer is A
Choice A reason:
The onset of warfarin is delayed, meaning it takes longer to achieve a therapeutic effect. Warfarin requires several days to adjust to effective anticoagulation levels because it works by inhibiting the synthesis of clotting factors that are already present and active in the bloodstream. In contrast, heparin has an immediate anticoagulant effect when administered, which is crucial for patients with acute conditions like deep vein thrombosis.
Choice B reason:
Warfarin does not prevent platelet aggregation. Instead, it works by inhibiting vitamin K-dependent clotting factors, which are essential for blood coagulation. Platelet aggregation is primarily inhibited by antiplatelet drugs like aspirin or clopidogrel, not anticoagulants like warfarin.
Choice C reason:
Heparin does not necessarily have fewer adverse effects compared to warfarin. Both anticoagulants have their specific risks and side effects. Heparin is preferred in acute settings due to its rapid onset of action, but it can cause complications like heparin-induced thrombocytopenia (HIT), which requires careful monitoring.
Choice D reason:
Heparin does not have a longer half-life than warfarin. In fact, heparin's half-life is relatively short, which is why it is often administered via continuous infusion or frequent subcutaneous injections. Warfarin has a longer half-life, but its delayed onset of action makes it less suitable for immediate anticoagulation needs.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A reason:
Arterial emboli from a heart valve are not the most common cause of myocardial ischemia. While emboli can obstruct blood flow and cause ischemia, they are a less common cause compared to atherosclerosis. Embolic events typically result from conditions like atrial fibrillation or valvular heart disease but do not account for the majority of myocardial ischemia cases.
Choice B reason:
Atherosclerosis is the most common cause of myocardial ischemia. It involves the buildup of plaque within the coronary arteries, leading to reduced blood flow and oxygen supply to the heart muscle. This condition is a leading cause of coronary artery disease and is responsible for the majority of myocardial ischemia cases.
Choice C reason:
Idiopathic vasospasm, although a cause of myocardial ischemia, is not as common as atherosclerosis. Vasospasm refers to the sudden constriction of a coronary artery, which can reduce blood flow to the heart muscle. However, it accounts for a smaller percentage of ischemia cases compared to the widespread prevalence of atherosclerosis.
Choice D reason:
Venous emboli are typically associated with conditions such as deep vein thrombosis and pulmonary embolism, affecting the venous circulation rather than the coronary arteries. They do not commonly cause myocardial ischemia, which primarily involves the coronary arteries and arterial blockages.
Correct Answer is A
Explanation
Choice A reason:
Taking an antacid one hour after ranitidine is recommended because antacids can affect the absorption of H2 receptor blockers like ranitidine. Ranitidine works by reducing stomach acid production, whereas antacids neutralize existing acid. Taking the antacid too close to the ranitidine can reduce the effectiveness of the ranitidine by altering the stomach's pH balance and affecting its absorption.
Choice B reason:
Taking an antacid and ranitidine at the same time does not enhance their effect. In fact, this can interfere with the absorption and effectiveness of ranitidine. Antacids can increase the pH of the stomach, which may reduce the absorption of ranitidine, thus diminishing its acid-reducing effects.
Choice C reason:
Taking both medications at the same time before meals is not advisable for the same reason as above. The simultaneous administration can reduce the effectiveness of ranitidine, as the increased pH caused by the antacid can interfere with the absorption of the H2 receptor blocker, thereby not providing the intended therapeutic effect.
Choice D reason:
The patient can take both medications, but they should be timed correctly to ensure optimal effectiveness. Saying that the patient needs to be on one medication only is incorrect. Both medications can be used together, but the antacid should be taken after ranitidine to avoid any interaction that might impair the effectiveness of ranitidine.
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