A patient was admitted to the intensive care unit with a diagnosis of acute myocardial infarction (MI) and is being treated for hypotension. The primary cause of hypotension is most likely:
decreased afterload due to vasodilation.
rapid heart rate.
decreased cardiac contractility, resulting in decreased cardiac output.
increased capillary permeability.
The Correct Answer is C
Choice A reason:
Decreased afterload due to vasodilation is not the primary cause of hypotension in acute myocardial infarction. While vasodilation can lower blood pressure, in the context of MI, the more direct and significant cause of hypotension is the reduced cardiac contractility leading to decreased cardiac output. Vasodilation and afterload reduction are not the primary mechanisms in this scenario.
Choice B reason:
A rapid heart rate, or tachycardia, can contribute to hypotension by reducing the time for the heart to fill with blood between beats. However, in the context of an acute myocardial infarction, the primary issue is the heart's ability to contract effectively. Therefore, while a rapid heart rate might exacerbate the condition, it is not the main cause of hypotension.
Choice C reason:
Decreased cardiac contractility, resulting in decreased cardiac output, is the primary cause of hypotension in acute myocardial infarction. The damaged heart muscle cannot pump effectively, leading to reduced blood flow and low blood pressure. This is a direct consequence of the myocardial damage caused by the infarction, making this the correct answer.
Choice D reason:
Increased capillary permeability is not a primary cause of hypotension in acute myocardial infarction. This condition is more associated with inflammatory responses, sepsis, or allergic reactions. The main cause of hypotension in MI is related to the heart's reduced pumping ability.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["A","B","D","E"]
Explanation
Choice A reason:
Nasogastric suctioning is a common intervention in managing acute pancreatitis when there is severe nausea, vomiting, or evidence of intestinal obstruction. The procedure helps to decompress the stomach, reduce pancreatic stimulation, and minimize the risk of aspiration. It is particularly indicated for patients who cannot tolerate oral intake or exhibit signs of paralytic ileus. By reducing gastric distension and suppressing pancreatic secretions, nasogastric suctioning aids in alleviating symptoms and improving patient outcomes.
Choice B reason:
Narcotic analgesics are essential for controlling the intense abdominal pain that accompanies acute pancreatitis. The pain arises due to inflammation and autodigestion of pancreatic tissue by enzymes like trypsin. Medications such as morphine or hydromorphone provide effective relief by acting on opioid receptors in the central nervous system. Adequate pain management is crucial not only for patient comfort but also to mitigate stress-related complications that can worsen inflammation or systemic effects.
Choice C reason:
Steroid therapy is generally not part of the treatment for acute pancreatitis unless there is an associated autoimmune component. In most cases, the use of steroids could exacerbate the condition or increase the risk of complications such as infections. As such, this option is not appropriate in routine management of acute pancreatitis.
Choice D reason:
Restriction of food intake is a critical component of the treatment plan. Fasting minimizes pancreatic stimulation and allows the inflamed pancreas to rest. Typically, patients are kept nil by mouth (NPO) until their symptoms subside. Nutrition can then be gradually reintroduced, starting with clear liquids and advancing as tolerated. Enteral feeding via a nasojejunal tube may be considered if prolonged fasting is required.
Choice E reason:
IV fluids are a cornerstone of acute pancreatitis management. Fluid resuscitation is necessary to address hypovolemia caused by fluid shifts, vomiting, and third-spacing of fluids into inflamed tissues. Aggressive hydration with isotonic crystalloids, such as normal saline or lactated Ringer's solution, helps maintain hemodynamic stability and improves microcirculation in the pancreas, reducing the risk of complications such as necrosis or organ failure.
Correct Answer is A
Explanation
Choice A reason:
In patients with chronic renal failure, the kidneys cannot effectively excrete magnesium, leading to the risk of hypermagnesemia. Magnesium hydroxide, commonly used as an antacid and laxative, should be used with caution or avoided in these patients to prevent magnesium accumulation in the blood, which can have serious cardiovascular and neuromuscular effects.
Choice B reason:
While cirrhosis affects liver function and can alter drug metabolism, magnesium hydroxide does not have a direct contraindication for use in patients with cirrhosis. However, caution is necessary due to potential electrolyte imbalances and altered pharmacokinetics. The primary concern with magnesium hydroxide in these patients is less critical compared to those with renal failure.
Choice C reason:
Hemorrhoids do not directly contraindicate the use of magnesium hydroxide. This condition primarily affects the veins around the rectum and anus and is unrelated to the systemic effects of magnesium. Therefore, the presence of hemorrhoids does not necessitate withholding the medication.
Choice D reason:
Undiagnosed abdominal pain is a significant consideration before administering magnesium hydroxide. The use of laxatives can exacerbate conditions like intestinal obstruction or acute abdomen. Therefore, caution is advised, but the immediate and more critical concern remains the risk posed to patients with chronic renal failure.
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