A patient with leukocytosis, fever and left lower quadrant (LLQ) pain will most likely have a diagnosis of

A. Hyperproteinemia and increased drug effect: In cirrhosis, hypoproteinemia (low protein levels) occurs due to decreased protein synthesis by the liver, and drug metabolism is often impaired, leading to increased drug effects, but hyperproteinemia is not a typical finding.

B. Hyperkalemia and fluid retention: While fluid retention is common due to hypoalbuminemia and portal hypertension, hyperkalemia is not a direct consequence of hepatocyte dysfunction.

C. Hypercortisolism and increased infection risk: Hypercortisolism is not typically associated with cirrhosis. However, increased infection risk is common due to compromised immune function.

D. An elevated blood glucose and ammonia level: In cirrhosis, the liver's ability to metabolize ammonia is impaired, leading to elevated levels. Additionally, impaired glucose metabolism can result in hyperglycemia.

A. Ischemia of the brain tissue is causing cellular injury, swelling, and malfunction of the contralateral side: Hemiparesis occurs on the side opposite to the brain lesion due to the crossing (decussation) of motor pathways.

B. The functioning of the right cranial nerve controlling facial movement is compromised by cerebral edema: While facial nerve involvement can cause drooping, the hemiparesis suggests a central (brain) rather than peripheral issue.

C. The sensorimotor tracts leading from the brain to the body decussate and control the arm and leg on the same side as the lesion: This is incorrect because the tracts control the contralateral side of the body.

D. The corticospinal tracts leading from the brain to the body decussate and control the ipsilateral arm and leg: This is incorrect; the corticospinal tracts control the contralateral side.