Heparin 10,000 units subcutaneous every 12 hours is ordered for a patient after a stent placement.
The heparin is available in a 20,000 units per 1 mL concentration.
How many milliliters would the nurse administer with each dose? (ROUND TO TENTHS; LABEL CORRECTLY TO RECEIVE CREDIT)
The Correct Answer is ["0.5"]
Step 1: The order is 10,000 units.
Step 2: The available concentration is 20,000 units per 1 mL.
Step 3: Calculate the volume to administer: 10,000 units ÷ (20,000 units ÷ 1 mL) = 0.5 mL. Answer: 0.5 mL.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["75"]
Explanation
Step 1 is 1.5 L × 1000 mL/L = 1500 mL.
Step 2 is 1500 mL ÷ 20 hours = 75 mL/hour.
Correct Answer is D
Explanation
Choice A rationale
Administering nitroglycerin on an as-needed basis for sudden, crushing chest pain is inappropriate because the immediate nature of the pain requires rapid intervention. Myocardial ischemia, often causing such pain, necessitates swift vasodilation to improve coronary blood flow and oxygen supply, preventing irreversible cardiac muscle damage. Delay can exacerbate ischemia.
Choice B rationale
Administering nitroglycerin on a prescribed schedule is typically reserved for prophylactic use in conditions like stable angina to prevent episodes, not for acute, sudden onset chest pain. This type of pain indicates an acute event, potentially myocardial infarction, demanding immediate action to mitigate cardiac damage through rapid vasodilation.
Choice C rationale
Administering medication at the earliest convenience is not appropriate for a STAT order in a patient experiencing sudden, crushing chest pain. "STAT" signifies an urgent need for the medication, indicating a life-threatening or rapidly evolving condition requiring immediate pharmacological intervention to avert significant physiological compromise.
Choice D rationale
Administering nitroglycerin immediately is crucial for sudden, crushing chest pain as it suggests acute myocardial ischemia or infarction. Nitroglycerin causes vasodilation, reducing preload and afterload, which decreases myocardial oxygen demand and improves coronary blood flow, thereby limiting cardiac damage and alleviating symptoms rapidly.
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