Myocardium is rigid and noncompliant, impeding ventricular filling and raising pressures during diastole is which form of cardiomyopathy?

A. We will have to drain the pus out of your pleural space: An empyema is characterized by the accumulation of pus in the pleural space, typically due to infection or inflammation. The primary treatment often involves draining the infected fluid to relieve symptoms and treat the underlying infection, making this statement accurate and appropriate for the patient.

B. These blebs in your lungs can rupture with exercise: This statement is not applicable to empyema. Blebs are associated with conditions like pulmonary bullae or emphysema, not empyema. Empyema specifically refers to pus in the pleural space, not blebs in the lungs.

C. You will be given a long course of antiviral medication: Antiviral medication is not typically indicated for empyema, as it is often caused by bacterial infections. Treatment usually involves antibiotics and drainage rather than antiviral therapy.

D. We will watch you for respiratory muscle fatigue: While monitoring for respiratory status is important in any patient with empyema, this statement does not specifically address the nature of empyema or its treatment. The focus should be on managing the infection and drainage of the pleural space.

A. Norepinephrine causes bronchial smooth muscle contraction and mucus secretion but it also causes high blood pressure: This statement is misleading in the context of asthma. Norepinephrine primarily acts on alpha and beta receptors, influencing blood pressure and bronchodilation but is not the main mediator in asthma pathophysiology. The focus should be on inflammation and airway responsiveness.

B. Uncontrolled inflammation leads to increased bronchial hyperresponsiveness and eventual scarring: This statement accurately describes the pathophysiologic process in asthma. Persistent inflammation in asthma can cause increased bronchial hyperresponsiveness, leading to airway narrowing and potential long-term remodeling and scarring of the airways if not controlled. Effective management is essential to prevent these adverse outcomes.

C. Immunoglobulin G causes smooth muscle contraction which will eventually weaken the respiratory muscles: Immunoglobulin E (IgE) is primarily involved in allergic reactions and asthma, and it does not directly cause smooth muscle contraction that weakens respiratory muscles.

D. The release of epinephrine leads to development of cardiac dysrhythmias: While epinephrine can have cardiovascular effects, including increased heart rate and potential for dysrhythmias, this is not directly relevant to the pathophysiology of asthma. The focus in asthma management is on controlling airway inflammation and bronchoconstriction rather than on cardiac issues.