Primary hypertension:
Is caused by hypertrophy and hyperplasia with associated fibrosis of the intima and media
Underactivity of the sympathetic nervous system and the RAAS
is caused by complicated interactions of genetics and the environment without evidence of a specific causative agent
is caused by systemic disease that raises peripheral vascular resistance and/or cardiac output
The Correct Answer is C
A. Is caused by hypertrophy and hyperplasia with associated fibrosis of the intima and media: While these changes can occur in the vascular walls over time due to hypertension, they are not the primary cause of primary hypertension itself. Primary hypertension is more complex and multifactorial.
B. Underactivity of the sympathetic nervous system and the RAAS: Primary hypertension is characterized by increased activity of the sympathetic nervous system and the renin-angiotensin-aldosterone system (RAAS), not underactivity. Increased activity of these systems contributes to elevated blood pressure.
C. Is caused by complicated interactions of genetics and the environment without evidence of a specific causative agent: This statement accurately describes primary hypertension, which results from a combination of genetic predisposition and environmental factors such as diet, physical activity, and stress. There is no single identifiable cause for primary hypertension, making this the correct answer.
D. Is caused by systemic disease that raises peripheral vascular resistance and/or cardiac output: This statement pertains more to secondary hypertension, which is due to identifiable causes such as kidney disease, endocrine disorders, or other systemic diseases. Primary hypertension does not arise from a specific systemic disease but rather from multifactorial influences.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A. It does not remain dormant but some host defenses can kill the bacteria: While the immune system can kill some of the Mycobacterium tuberculosis bacilli, in many cases, the bacteria evade complete eradication and persist in a dormant state within the lungs. This does not mean the infection is fully eliminated, as it can reactivate later.
B. Virulence factors in the bacilli weaken over time, leading to apoptosis: Mycobacterium tuberculosis does not naturally lose its virulence over time. Instead, it can persist in a latent state due to immune containment, not because of a reduction in its ability to cause disease.
C. The bacilli can become isolated within tubercles in the lungs, possibly encapsulated: In latent tuberculosis, the immune system walls off the bacilli within granulomas (tubercles), preventing active disease. These granulomas may be encapsulated with fibrous tissue, restricting bacterial growth and spread. The bacteria remain dormant but can reactivate if the immune system weakens.
D. Macrophages attack and phagocytize new areas of infection: While macrophages do play a role in the immune response to tuberculosis, they are often unable to completely eradicate the bacilli. Instead, the bacteria can survive within macrophages and trigger the formation of granulomas, which help contain but not eliminate the infection.
Correct Answer is C
Explanation
A. Triglycerides: Elevated triglyceride levels are associated with an increased risk of atherosclerosis and cardiovascular disease. High triglycerides can contribute to the buildup of plaque in the arteries, leading to a higher risk of heart disease and complications such as heart attacks or strokes.
B. Very low density lipoproteins (VLDLs): Elevated VLDL levels are considered harmful and are linked to a higher risk of atherosclerosis. VLDLs contribute to the accumulation of triglycerides and promote the development of plaque in the arteries, which can significantly increase cardiovascular risk over time.
C. High density lipoproteins (HDLs): Elevated HDL levels are considered protective against the development of atherosclerosis. HDLs help transport cholesterol away from the arteries and back to the liver for excretion, which not only reduces the risk of plaque buildup but also lowers the likelihood of heart disease and enhances overall cardiovascular health.
D. Low density lipoproteins (LDLs): Elevated LDL levels are known to be a major risk factor for atherosclerosis. LDL cholesterol can contribute to the formation of plaques in the arteries, increasing the risk of heart disease and stroke, and it is important to manage LDL levels for better cardiovascular health.
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