Necrotic death of brain tissue usually produces:

Choice A Reason:

Fat necrosis occurs when fatty tissues are damaged, leading to the release of enzymes that break down fat cells. This type of necrosis is commonly seen in the pancreas and breast tissue, often due to trauma or pancreatitis. It is not typically associated with brain tissue.

Choice B Reason:

Coagulative necrosis is characterized by the preservation of the basic outline of the coagulated cells for a few days after cell death. This type of necrosis is usually seen in tissues affected by ischemia, such as the heart, kidneys, and adrenal glands. However, it is not the typical pattern of necrosis seen in brain tissue.

Choice C Reason:

Caseous necrosis is a form of cell death in which the tissue maintains a cheese-like appearance. It is most commonly associated with tuberculosis infections in the lungs. This type of necrosis is not typically seen in brain tissue.

Choice D Reason:

Liquefactive necrosis is the correct answer. This type of necrosis occurs when the tissue becomes soft and liquefied, often forming a pus-filled cavity. It is commonly seen in the brain due to ischemic injury or bacterial infections. The high lipid content and low structural support in brain tissue make it particularly susceptible to liquefactive necrosis.

Choice A Reason:

Beta2 agonists, such as terbutaline, are commonly used to manage premature labor. These drugs work by stimulating beta2 adrenergic receptors in the smooth muscles of the uterus, leading to relaxation and suppression of contractions. Therefore, questioning the use of a beta2 agonist in this scenario would not be appropriate.

Choice B Reason:

Anticholinergic drugs are not typically used to manage premature labor. They work by blocking the action of acetylcholine in the nervous system, which can lead to various effects such as reduced secretions and relaxation of smooth muscles. However, they are not specifically indicated for stopping uterine contractions, so questioning their use in this context is less relevant.

Choice C Reason:

This is the correct answer. Beta2 antagonists, also known as beta-blockers, work by blocking beta2 adrenergic receptors, which can lead to increased uterine contractions. Using a beta2 antagonist in a patient experiencing premature labor would be counterproductive and could exacerbate the condition. Therefore, it is appropriate to question the use of beta2 antagonists in this scenario.

Choice D Reason:

Alpha1 agonists are not typically used in the management of premature labor. These drugs stimulate alpha1 adrenergic receptors, leading to vasoconstriction and increased blood pressure. They do not have a direct effect on uterine contractions, so questioning their use in this context is less relevant.