Recognizing the role of the kidney in the production of erythropoietin, the nurse assesses a client with chronic kidney disease (CKD) for which clinical manifestation?
Pallor.
Petechiae.
Jaundice.
Pruritus.
The Correct Answer is A
Choice A reason: In CKD, impaired kidneys produce less erythropoietin, reducing red blood cell production and causing anemia. Pallor results from decreased hemoglobin, a hallmark of CKD-related anemia. This manifestation aligns with the kidney’s role in erythropoiesis, making it the primary clinical sign the nurse should assess in this client.
Choice B reason: Petechiae, small skin hemorrhages, result from platelet dysfunction or vascular issues, not directly from reduced erythropoietin in CKD. While CKD may cause uremic bleeding tendencies, petechiae are less specific than pallor, which directly reflects anemia due to impaired erythropoietin production, a core pathophysiological feature.
Choice C reason: Jaundice, caused by bilirubin accumulation, indicates liver dysfunction or hemolysis, not erythropoietin deficiency. CKD does not typically cause jaundice unless complicated by unrelated conditions. Pallor from anemia is a more direct consequence of reduced erythropoietin, making it the priority manifestation for assessment in CKD.
Choice D reason: Pruritus in CKD results from uremic toxin accumulation or calcium-phosphate imbalances, not erythropoietin deficiency. While common, it is unrelated to the kidney’s erythropoiesis role. Pallor, linked to anemia from low erythropoietin, is the most relevant clinical sign for the nurse to assess in this context.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Choice A reason: Polyuria, excessive urination, is not caused by renal calculi movement. Stones obstruct the ureter, reducing urine flow and causing pain, not increased output. Polyuria is associated with conditions like diabetes, making this incorrect for the pathophysiological change linked to calculi movement in the urinary tract.
Choice B reason: Uric acid increases may contribute to stone formation but are not a change caused by calculi movement. Movement triggers pain and obstruction, not serum uric acid changes. Renal colic is the direct result of stones moving, making this choice incorrect for the pathophysiological effect.
Choice C reason: Cystitis, bladder inflammation, may occur secondary to stones but is not the primary change from calculi movement. Stones moving through the ureter cause renal colic due to obstruction and spasm. Cystitis is a complication, not the direct pathophysiological change, making this incorrect.
Choice D reason: Renal colic, severe pain from ureteral obstruction and smooth muscle spasm, occurs as renal calculi move through the urinary tract. Stones irritate and block the ureter, triggering intense, colicky pain. This is the primary pathophysiological change, aligning with urological evidence for stone movement effects.
Correct Answer is D
Explanation
Choice A reason: Normal antidiuretic hormone levels regulate water balance but do not address thirst in diabetes mellitus, which is caused by hyperglycemia-induced osmotic diuresis. Controlling blood glucose corrects the osmotic imbalance driving thirst, making ADH maintenance less relevant and incorrect for this manifestation’s control.
Choice B reason: Increasing serum osmolarity would worsen thirst, as high osmolarity from hyperglycemia causes dehydration and polydipsia. The goal is to reduce osmolarity by controlling blood glucose, which mitigates osmotic diuresis. This choice is incorrect, as it exacerbates the mechanism driving the client’s symptom.
Choice C reason: Increased acetone excretion occurs in diabetic ketoacidosis, not directly related to thirst in new diabetes mellitus. Thirst results from hyperglycemia causing osmotic diuresis. Controlling glucose levels addresses the root cause, making acetone excretion irrelevant and incorrect for managing polydipsia in this client.
Choice D reason: Increased thirst in diabetes mellitus results from hyperglycemia causing osmotic diuresis, leading to dehydration. Controlling blood glucose levels reduces serum osmolarity, preventing fluid loss and alleviating thirst. This is the primary physiologic mechanism, supported by endocrinology evidence for managing diabetes-related polydipsia effectively.
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