The labia minora and labia majora are parts of which aspect of the female anatomy?
Uterus
Fallopian tube
Vulva
Ovary
The Correct Answer is C
A. Uterus: The uterus is an internal reproductive organ (the womb) and does not include the labia.
B. Fallopian tube: Fallopian (uterine) tubes are internal conduits for the oocyte; they are not the labia.
C. Vulva: The vulva is the external female genitalia and includes the labia majora and labia minora .
D. Ovary: The ovaries are internal gonads that produce oocytes and hormones; they are not the labia.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A. Proximal convoluted tubule: The PCT reabsorbs the majority of filtered water and solutes (≈65% Na⁺/H₂O, all filtered glucose and amino acids, many ions).
B. Efferent arteriole: An efferent arteriole is a blood vessel leaving the glomerulus (carries blood), not a tubular segment that reabsorbs filtrate.
C. Afferent arteriole: Afferent arteriole supplies blood to the glomerulus; it is not involved in tubular reabsorption.
D. Loop of Henle: The loop helps concentrate urine (water reabsorption in the descending limb, ion transport in the ascending limb) but the PCT is the primary site for bulk reabsorption.
Correct Answer is ["A","B","D"]
Explanation
A. It promotes vasoconstriction of blood vessels:Angiotensin II (RAAS mediator) is a potent vasoconstrictor, raising systemic vascular resistance and affecting renal blood flow.
B. It stimulates thirst:Angiotensin II acts on the brain (hypothalamus) to stimulate thirst and ADH release, promoting fluid intake and retention.
C. It lowers blood pressure:RAAS activation raises blood pressure by vasoconstriction and increased sodium/water retention.
D. It increases water reabsorption in the kidneys:Aldosterone promotes Na⁺ reabsorption (water follows), and angiotensin II/ADH actions lead to increased water retention—overall increasing water reabsorption.
E. It stimulates the release of renin:Renin release is the initiating step of RAAS activation (triggered by low renal perfusion); once RAAS is active, angiotensin II typically provides negative feedback that suppresses further renin release rather than stimulating it.
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