What are the possible physiological changes in the brain of a client diagnosed with Alzheimer's disease? (Select all that apply)
Brain atrophy
Overabundance of plaques (amyloid beta)
Overabundance of tangles (tau protein)
Enlargement of the hippocampus
Enlarged cerebral cortex
Correct Answer : A,B,C
A. Brain atrophy is a common physiological change in the brain of individuals with Alzheimer's disease.
B. An overabundance of plaques, specifically amyloid beta, is a characteristic feature of Alzheimer's disease.
C. An overabundance of tangles, specifically tau protein, is also a characteristic feature of Alzheimer's disease.
D. Enlargement of the hippocampus is not typically associated with Alzheimer's disease; rather, it tends to shrink.
E. Enlarged cerebral cortex is not a typical physiological change in Alzheimer's disease; it actually tends to shrink.
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Related Questions
Correct Answer is D
Explanation
A. This statement may cause unnecessary distress to the client and is not necessarily true in this situation.
B. While the confusion may be temporary, this statement may not provide enough reassurance or information.
C. This statement may not be accurate or helpful in addressing the client's concerns about confusion.
D. This statement provides reassurance and offers a positive outlook, indicating that the confusion is likely to improve as the client's condition gets better.
Correct Answer is C
Explanation
A) Incorrect. Thyroxine is a hormone produced by the thyroid gland and is not directly implicated in the etiology of schizophrenia.
B) Incorrect. Erythropoietin is a hormone that stimulates the production of red blood cells and is not directly implicated in the etiology of schizophrenia.
C) Correct. Glutamate, an excitatory neurotransmitter, has been implicated in the development of schizophrenia. Abnormalities in glutamate signaling have been identified in individuals with schizophrenia.
D) Incorrect. While serotonin abnormalities have been associated with mood disorders such as depression, they are not considered a primary factor in the etiology of schizophrenia.
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