Which is the hormone that stimulates the release of aldosterone from the adrenal cortex.
Renin.
Angiotensin I.
Angiotensin II.
Antidiuretic hormone (ADH).
The Correct Answer is C
Angiotensin II is a hormone that stimulates the adrenal cortex to release aldosterone. Aldosterone is a hormone that helps regulate blood pressure by increasing the reabsorption of sodium and water and the excretion of potassium by the kidneys.
Choice A is wrong because renin is not a hormone but an enzyme that catalyzes the conversion of angiotensinogen to angiotensin I1.
Choice B is wrong because angiotensin I is an inactive precursor of angiotensin II that is converted by angiotensin-converting enzyme (ACE) in the lungs.
Choice D is wrong because antidiuretic hormone (ADH) is a hormone that regulates water balance by increasing the reabsorption of water by the kidneys, but it does not affect aldosterone secretion.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Acute renal injury (ARI) is a term for a reversible syndrome that results in decreased glomerular filtration rate (GFR) and oliguria. GFR is a measure of how well the kidneys filter blood and oliguria is a condition of producing less than normal amounts of urine.
Choice B is wrong because chronic renal injury (CRI) is not a reversible syndrome, but a progressive loss of kidney function over months or years.
Choice C is wrong because end-stage renal disease (ESRD) is not a reversible syndrome, but a condition where the kidneys have lost most or all of their function and dialysis or transplantation is required.
Choice D is wrong because acute tubular necrosis (ATN) is not a term for a syndrome, but a specific type of acute kidney injury that involves damage to the tubules, the part of the nephron that reabsorbs water and solutes from the filtrate.
Correct Answer is C
Explanation
This is because intravenous potassium supplementation is indicated for patients with profound hypokalemia (plasma K+ <2.5 mmol/L) or cardiac arrhythmia. The rate of infusion should not exceed 10 mmol/hour to prevent complications such as hyperkalemia, cardiac arrhythmias, and phlebitis.
Choice A is wrong because monitoring urine output every 8 hours is not sufficient to prevent complications from intravenous potassium replacement therapy.
Urine output should be monitored more frequently (at least every 4 hours) to assess renal function and fluid balance.
Choice B is wrong because administering potassium via a bolus injection is dangerous and can cause fatal cardiac arrhythmias.
Potassium should never be given by intravenous push or intramuscular injection.
Choice D is wrong because encouraging the client to eat potassium-rich foods is not appropriate for patients receiving intravenous potassium replacement therapy.
Oral potassium supplementation is preferred for patients with mild to moderate hypokalemia (plasma K+ 2.5-3.5 mmol/L) who can eat and absorb oral potassium.
Potassium-rich foods include potatoes, legumes, juices, seafood, leafy greens, dairy, tomatoes and bananas.
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