A client is receiving anticoagulant therapy to prevent blood clots. The nurse identifies a potential adverse effect of anticoagulation as:
Increased risk of bleeding.
Elevated blood pressure readings.
Hyperactivity and restlessness.
Increased blood viscosity.
The Correct Answer is A
A) Correct. Anticoagulant therapy prevents blood from clotting effectively, which can lead to an increased risk of bleeding.
B) Incorrect. Anticoagulant therapy is not associated with elevated blood pressure readings.
C) Incorrect. Hyperactivity and restlessness are not typical adverse effects of anticoagulant therapy.
D) Incorrect. Anticoagulant therapy does not increase blood viscosity (thickness); instead, it reduces the blood's ability to form clots.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A) Incorrect. Hemophilia does not increase the risk of forming blood clots. In fact, it causes difficulty in forming blood clots, leading to an increased risk of bleeding and hemorrhage.
B) Incorrect. Platelet count is not directly affected in hemophilia. It is primarily a disorder of clotting factors.
C) Correct. Hemophilia is a genetic disorder characterized by a deficiency or absence of specific clotting factors, which impairs the blood's ability to clot properly.
D) Incorrect. Hemophilia does not affect the number of red blood cells, which are responsible for oxygen transport.
Correct Answer is B
Explanation
A) Incorrect. Bradycardia is not typically an early sign of compensatory response to hypovolemia. The body often responds to hypovolemia with an increased heart rate (tachycardia).
B) Correct. In the early stages of hypovolemia, the body may attempt to compensate by increasing blood pressure to maintain perfusion to vital organs.
C) Incorrect. Warm, flushed skin may be present in the initial stages of hypovolemia, but it is not necessarily an early sign of compensatory response.
D) Incorrect. Decreased urine output is a later sign of hypovolemia when the body's compensatory mechanisms are no longer effective in maintaining blood flow to the kidneys.
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