A client taking diltiazem, a calcium channel blocker, for hypertension has come to the clinic for a follow-up appointment. The nurse will assess the client for which adverse effects?
Chest pain and pale skin
Shortness of breath and wheezing
Peripheral edema and bradycardia
Tachycardia and anxiety
The Correct Answer is C
A. Chest pain and pale skin are not typical adverse effects associated with diltiazem use.
B. Shortness of breath and wheezing may occur due to other conditions or medications but are not common adverse effects of diltiazem.
C. Calcium channel blockers like diltiazem can cause peripheral edema due to vasodilation and bradycardia as a result of their action on the heart rate. Monitoring for these effects is essential in patients taking diltiazem.
D. Tachycardia is not an expected adverse effect of diltiazem; in fact, it is used to manage conditions where tachycardia may be present. Anxiety is also not a direct adverse effect of this medication.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
A. Sodium levels are important to monitor, but they are not the primary concern with furosemide treatment.
B. Bone marrow function is not a direct concern related to furosemide therapy.
C. Calcium levels are less critical compared to potassium when monitoring a client on furosemide.
D. Potassium levels are correct because furosemide is a loop diuretic that can lead to significant potassium loss, putting the client at risk for hypokalemia, which can have serious cardiac implications.
Correct Answer is C
Explanation
A. Short-acting insulin does not cover basal needs; that is the role of long-acting insulin.
B. Intermediate-acting insulin does not primarily cover mealtime glucose spikes; it provides a more prolonged effect.
C. Short-acting insulin is used to manage blood glucose during meals, while intermediate-acting insulin helps maintain glucose control between meals and overnight, making this the correct choice.
D. Short-acting and intermediate-acting insulins have different onset and peak times; they do not share the same pharmacokinetic properties.
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