A nurse states, "I evaluated a new patient using the modified SAD PERSONS scale and got a score of 10. Should I send the patient home?" Select the best reply.
A score over 8 requires immediate hospitalization.
Give the patient a follow-up appointment. Hospitalization may be needed soon.
That action would seem to be appropriate based on the score.
I think you should strongly consider a benzodiazepine for this patient.
The Correct Answer is A
Choice A reason: A SAD PERSONS score over 8 indicates high suicide risk, driven by serotonin dysregulation and amygdala hyperactivity, necessitating hospitalization to prevent self-harm. Immediate intervention stabilizes neurochemical imbalances and ensures safety, addressing the acute risk effectively.
Choice B reason: A follow-up appointment is inadequate for a score of 10, indicating high suicide risk. Serotonin and dopamine dysregulation heighten impulsivity, and delaying hospitalization risks self-harm, as outpatient care cannot address acute amygdala-driven suicidal ideation effectively.
Choice C reason: Sending the patient home with a score of 10 is unsafe, as high suicide risk involves serotonin deficits and amygdala hyperactivity. This disregards the need for immediate intervention to stabilize neurochemical imbalances and ensure safety, making it inappropriate.
Choice D reason: Benzodiazepines address anxiety but not suicide risk directly. A score of 10 indicates severe serotonin dysregulation and amygdala-driven impulsivity, requiring hospitalization, not anxiolytics, which may mask symptoms without addressing the underlying neurochemical suicide risk.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Choice A reason: Neuroleptic malignant syndrome is associated with antipsychotics, not SSRIs like paroxetine, causing muscle rigidity and hyperthermia via dopamine blockade. The client’s symptoms, including hyperreflexia and diarrhea, align with serotonin excess, not dopamine-related issues, making this condition unlikely.
Choice B reason: Agranulocytosis, a severe reduction in white blood cells, is unrelated to paroxetine’s mechanism. SSRIs increase serotonin, not affecting hematopoiesis. The client’s symptoms like hyperpyrexia and hyperreflexia indicate serotonin toxicity, not an immunological or bone marrow disorder.
Choice C reason: Acute dystonic reactions involve muscle spasms from antipsychotics’ dopamine antagonism, not SSRIs. Paroxetine’s serotonin increase causes hyperreflexia and hyperpyrexia, consistent with serotonin syndrome, not extrapyramidal symptoms, making this diagnosis inappropriate for the described clinical presentation.
Choice D reason: Serotonin syndrome results from excessive serotonin due to paroxetine, an SSRI, overstimulating 5-HT receptors, causing hyperreflexia, hyperpyrexia, and autonomic instability. These symptoms reflect serotonin-driven neural excitation, particularly in the brainstem and spinal cord, matching the client’s clinical presentation accurately.
Correct Answer is A
Explanation
Choice A reason: Bupropion is an atypical antidepressant, inhibiting dopamine and norepinephrine reuptake, enhancing prefrontal cortex and reward system activity. Unlike SSRIs or MAOIs, it minimally affects serotonin, targeting catecholamines to improve mood and energy, aligning with its unique mechanism in treating depression.
Choice B reason: Tricyclic antidepressants block serotonin and norepinephrine reuptake and have anticholinergic effects, unlike bupropion, which targets dopamine and norepinephrine without significant anticholinergic activity. This distinct pharmacological profile excludes bupropion from the tricyclic class, making this choice incorrect.
Choice C reason: Selective serotonin reuptake inhibitors like fluoxetine target serotonin exclusively, increasing 5-HT levels. Bupropion primarily inhibits dopamine and norepinephrine reuptake, with minimal serotonin effects, making it an atypical antidepressant, not an SSRI, due to its distinct neurochemical action.
Choice D reason: Monoamine oxidase inhibitors like phenelzine prevent monoamine breakdown, increasing serotonin, dopamine, and norepinephrine. Bupropion’s selective reuptake inhibition of dopamine and norepinephrine, without MAO inhibition, distinguishes it as an atypical antidepressant, not an MAOI, due to its specific mechanism.
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