Abnormal red blood cell counts have what consequences on health?
Altered oxygen-carrying capability of blood
Altered ability to clot blood
Altered ability to fight infection
Altered heart rate and contractility
The Correct Answer is A
A. Altered oxygen-carrying capability of blood: RBCs (via hemoglobin) are the primary carriers of oxygen; too few (anemia) or dysfunctional RBCs reduce O₂ delivery, too many (polycythemia) can alter flow.
B. Altered ability to clot blood: clotting is primarily mediated by platelets and clotting factors, not by RBC count (RBCs can influence viscosity but are not the main clotting elements).
C. Altered ability to fight infection: fighting infection is mainly the role of leukocytes (WBCs), not RBCs.
D. Altered heart rate and contractility: significant changes in RBC number (especially anemia) can cause compensatory increases in heart rate and contractility to maintain oxygen delivery; polycythemia can also change cardiac workload.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
A. arteriole: arterioles are small, muscular arteries with thicker walls and no valves; lymphatics are thin-walled and valved.
B. artery: arteries have thick, muscular, elastic walls to withstand high pressure; lymphatic vessels are thin-walled and low-pressure.
C. capillary: capillaries are tiny single-cell-thick vessels for exchange; lymphatics are larger and have valves.
D. vein: lymphatic vessels have thin walls and valves and operate under low pressure, making them most similar in structure/function to veins.
Correct Answer is B
Explanation
A. Converts fibrinogen to fibrin: that reaction is carried out by thrombin, not plasmin.
B. Breaks down fibrin, dissolving the clot: plasmin is the primary enzyme of fibrinolysis, cleaving fibrin to dissolve formed clots.
C. Triggers the extrinsic clotting mechanism, leading to clot formation: the extrinsic pathway is initiated by tissue factor (factor III) interacting with factor VII, not plasmin.
D. Inactivates thrombin, slowing clot formation: plasmin’s main role is fibrin breakdown; thrombin is primarily regulated by antithrombin, thrombomodulin–protein C systems. (Plasmin can degrade some clotting proteins but it is not described as the main inactivator of thrombin.)
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