An older patient with cardiogenic shock is cool and clammy. Hemodynamic monitoring indicates a high systemic vascular resistance (SVR). Which intervention should the nurse anticipate?
Increase the rate for the dopamine infusion.
Increase the rate for the sodium nitroprusside infusion.
Decrease the rate for the nitroglycerin infusion.
Decrease the rate for the 5% dextrose in normal saline infusion.
The Correct Answer is B
B. Sodium nitroprusside is a potent vasodilator used to reduce systemic vascular resistance and afterload, thereby improving cardiac output and tissue perfusion in cardiogenic shock. In this scenario, where the patient has cool and clammy skin with high SVR, indicating peripheral vasoconstriction,
increasing the rate of sodium nitroprusside infusion can help vasodilate peripheral vessels, reduce afterload, and improve tissue perfusion
A. Dopamine is a medication commonly used in the management of cardiogenic shock to increase cardiac output and systemic blood pressure. However, in this scenario where the patient is cool and clammy with high SVR, indicating vasoconstriction and potential peripheral hypoperfusion, increasing the rate of dopamine infusion may further increase systemic vascular resistance and exacerbate peripheral vasoconstriction. This can worsen tissue perfusion and exacerbate the patient's condition.
.
C. Nitroglycerin is another vasodilator commonly used in the management of cardiogenic shock to reduce preload and afterload, thereby improving cardiac output and tissue perfusion. However, decreasing the rate of nitroglycerin infusion may further exacerbate vasoconstriction and increase SVR, worsening tissue perfusion in this scenario. Therefore, decreasing the rate of nitroglycerin infusion is not indicated.
D. Intravenous fluids such as 5% dextrose in normal saline are typically administered to maintain adequate intravascular volume and perfusion pressure in shock states. However, decreasing the rate of intravenous fluid infusion may further decrease intravascular volume and preload, potentially exacerbating hypoperfusion and worsening the patient's condition. Therefore, decreasing the rate of intravenous fluid infusion is not indicated in this scenario.
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Related Questions
Correct Answer is D
Explanation
D. Myocardial infarction (heart attack) is a common cause of cardiogenic shock. In myocardial infarction, part of the heart muscle becomes ischemic or necrotic due to occlusion of a coronary artery. This leads to impaired cardiac function and reduced cardiac output, resulting in cardiogenic shock. Prompt recognition and treatment of myocardial infarction are crucial to prevent or manage cardiogenic shock.
A. Anaphylaxis is a severe allergic reaction that can lead to systemic vasodilation and distributive shock, but it is not a common cause of cardiogenic shock. In anaphylaxis, the primary mechanism of shock is typically related to widespread vasodilation and increased vascular permeability rather than impaired cardiac function.
B. Hypovolemic shock occurs due to a decrease in intravascular volume, leading to inadequate tissue perfusion. It is not a common cause of cardiogenic shock, as the underlying mechanism is different. In hypovolemic shock, the primary issue is the loss of circulating blood volume, whereas cardiogenic shock involves impaired cardiac function.
C. Pulmonary embolism can lead to acute right heart strain or failure, which may result in hemodynamic instability and shock. However, pulmonary embolism typically causes obstructive shock rather than cardiogenic shock. Obstructive shock occurs when blood flow is obstructed, such as by a pulmonary embolism, leading to reduced cardiac output.
Correct Answer is B
Explanation
B. Cheyne-Stokes respirations involve a cyclical pattern of breathing characterized by gradual increase and decrease in the depth and rate of respirations, with periods of hyperventilation alternating with periods of apnea. It is commonly seen in patients with neurological disorders, heart failure, or drug overdose.
A. Apneustic respirations are characterized by prolonged inspiratory gasps followed by a brief pause and insufficient expiration. This pattern is often associated with damage to the pons in the brainstem.
C. Stridor is a high-pitched, noisy respiratory sound caused by turbulent airflow through partially obstructed airways. It is typically heard during inspiration and is often associated with upper airway obstruction, such as in cases of croup or epiglottitis.
D. Kussmaul respirations are deep, rapid, and labored breathing patterns often seen in patients with metabolic acidosis, particularly diabetic ketoacidosis. Unlike Cheyne-Stokes respirations, Kussmaul respirations do not involve periods of apnea.
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