Neuron that use Acetylcholine (ACh) as its neurotransmitter are what type of neuron?
GABA-ergic
Dopaminergic
Cholinergic
Serotonergic
The Correct Answer is C
A) GABA-ergic:
GABA-ergic neurons use gamma-aminobutyric acid (GABA) as their neurotransmitter, not acetylcholine (ACh). GABA is the main inhibitory neurotransmitter in the brain, playing a crucial role in reducing neuronal excitability.
B) Dopaminergic:
Dopaminergic neurons release dopamine as their neurotransmitter. Dopamine is involved in several critical functions, including movement, reward, and regulation of mood. Since acetylcholine is not involved in dopaminergic transmission, this is not the correct answer.
C) Cholinergic:
Cholinergic neurons use acetylcholine (ACh) as their neurotransmitter. These neurons are involved in many functions, including muscle activation (in the somatic nervous system), as well as modulating parasympathetic responses in the autonomic nervous system.
D) Serotonergic:
Serotonergic neurons release serotonin (5-HT), which is a neurotransmitter involved in regulating mood, appetite, sleep, and other functions. Acetylcholine is not involved in serotonergic transmission
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Related Questions
Correct Answer is C
Explanation
A) Norepinephrine (Levophed): Norepinephrine is a potent vasoconstrictor primarily used in acute hypotensive states to raise blood pressure. It is not indicated for the treatment of seasonal rhinitis, as its primary effect is not on the nasal passages or the symptoms associated with allergies, such as congestion.
B) Dopamine (Intropin): Dopamine is typically used in critical care settings for conditions such as shock and low blood pressure. While it affects dopaminergic and adrenergic receptors to improve renal perfusion and increase heart rate, it is not used to treat conditions like seasonal rhinitis, which involve nasal congestion.
C) Ephedrine (generic): Ephedrine is a sympathomimetic drug that stimulates alpha-adrenergic receptors, leading to vasoconstriction in the nasal passages and a reduction in swelling and congestion. This makes it a suitable choice for managing the symptoms of seasonal rhinitis, as it helps relieve nasal congestion by constricting blood vessels in the nasal mucosa.
D) Dobutamine (Dobutrex): Dobutamine is primarily used in the treatment of heart failure and shock because it increases heart rate and cardiac output. It does not have the properties needed to alleviate nasal congestion in rhinitis and is therefore not appropriate for this condition.
Correct Answer is C
Explanation
A) Hypothalamus and the medulla: While the hypothalamus and medulla play critical roles in regulating autonomic functions and overall sympathetic nervous system activity, the primary origin of the sympathetic nervous system's neural impulses comes from the spinal cord, specifically in the thoracic and lumbar regions. The hypothalamus and medulla are involved in coordinating and regulating sympathetic activity rather than being the origin of the impulses themselves.
B) Cranium and sacral area of the spinal cord: The cranium and sacral regions are primarily associated with the parasympathetic nervous system, not the sympathetic nervous system. The parasympathetic nervous system's nerve fibers arise from the brainstem and the sacral region, while the sympathetic fibers originate from the thoracic and lumbar areas.
C) Thoracic and lumbar section of the spinal cord: The sympathetic nervous system originates in the thoracolumbar region of the spinal cord, which includes the thoracic and lumbar segments (T1-L2). These regions house the preganglionic neurons whose axons exit the spinal cord and synapse in sympathetic ganglia, leading to the sympathetic effects on organs and tissues. This makes the thoracic and lumbar sections the correct location for the origin of SNS impulses.
D) Nerve membrane: The nerve membrane, or the cellular membrane of individual neurons, is not the location where impulses originate. The origin of the impulses is in the central nervous system (CNS), specifically in the spinal cord for the sympathetic system, not at the level of the individual nerve membranes.
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