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Select if the routes of administration listed below are enteral or parenteral.

A.

Subcutaneous

B.

NG Tube

C.

Oral

D.

Rectal

E.

Intramuscular

F.

Intravenous

Answer and Explanation

The Correct Answer is {"A":{"answers":"B"},"B":{"answers":"A"},"C":{"answers":"A"},"D":{"answers":"A"},"E":{"answers":"B"},"F":{"answers":"B"}}

  • Subcutaneous: A parenteral route where medication is injected into the tissue layer between the skin and muscle. It bypasses the gastrointestinal (GI) tract, allowing for faster absorption of certain medications like insulin or heparin.
  • NG Tube: A form of enteral administration that delivers medication directly into the stomach via a nasogastric tube. This route utilizes the GI tract and is often used for patients who cannot swallow.
  • Oral: A classic enteral route involving medication taken by mouth and absorbed through the digestive tract. It is the most common and convenient method of drug delivery.
  • Rectal: An enteral route that involves administration of medication into the rectum, where it is absorbed by the lower GI tract. It is often used when oral administration is not feasible.
  • Intramuscular: A parenteral route where medication is injected into a muscle, offering faster absorption than enteral routes. Common for vaccines and certain pain medications.
  • Intravenous: A parenteral route that delivers medication directly into the bloodstream. It provides the most rapid onset of action and is used for critical care, emergencies, and precise dosing.


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Related Questions

Correct Answer is ["150"]

Explanation

Total volume to be infused = 1200 mL

Total infusion time = 8 hours

  • Calculate the flow rate in mL per hour (mL/hr):

Flow rate (mL/hr) = Total volume (mL) / Total infusion time (hours)

= 1200 mL / 8 hours

= 150 mL/hr

Correct Answer is B

Explanation

A. Cardiotoxicity: While NSAIDs can contribute to cardiovascular risks such as hypertension and fluid retention, nephrotoxicity is the more direct explanation for the elevated kidney function tests and potassium level. Cardiotoxicity typically presents with symptoms like heart failure or arrhythmias rather than kidney-related lab abnormalities.
B. Nephrotoxicity: NSAIDs inhibit prostaglandin synthesis, which plays a key role in maintaining renal blood flow, especially in older adults or those with preexisting renal impairment. Chronic NSAID use can reduce glomerular filtration, leading to elevated creatinine, hyperkalemia, and worsening blood pressure control, all of which are evident in this patient.
C. Neurotoxicity: Neurotoxic effects are not commonly associated with NSAID use. Symptoms of neurotoxicity include confusion or seizures, which are not relevant to the clinical findings in this scenario.
D. Hepatotoxicity: NSAID-induced liver injury is rare and would typically present with elevated liver enzymes (AST, ALT), not elevated creatinine or potassium. The current lab findings and blood pressure changes are more consistent with kidney involvement.

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