The health care provider recommends daily fiber supplements for an elderly client who is experiencing frequent constipation. What statement is important for the nurse to include when educating a client about these supplements?
Bulk-forming agents decrease the absorption of nutrients in the intestines so you will need a daily vitamin
Fiber can exacerbate your constipation if you do not drink at least 8 glasses of water daily
If you take fiber long term, you can become dependent on it to have a bowel movement
Your bowel regimen will improve if you delay responding to your urge to defecate
The Correct Answer is B
Choice A reason: Bulk-forming fiber supplements, like psyllium, increase stool bulk without significantly decreasing nutrient absorption. While high doses may slightly affect mineral uptake, routine vitamin supplementation is not required. This statement is inaccurate, as nutrient malabsorption is not a primary concern with fiber supplements.
Choice B reason: Fiber supplements require adequate hydration (at least 8 glasses of water daily) to swell and soften stool, promoting bowel movements. Insufficient water can cause fiber to harden, worsening constipation. This statement is accurate, as hydration is critical for the efficacy and safety of fiber supplements.
Choice C reason: Long-term fiber use does not cause dependency; it mimics natural dietary fiber, promoting regular bowel movements. The colon adapts to increased bulk without losing intrinsic motility. This statement is inaccurate, as fiber supports, not undermines, normal bowel function in chronic use.
Choice D reason: Delaying the urge to defecate can worsen constipation by causing stool to harden and reducing rectal sensitivity. Prompt response to bowel urges promotes regularity. This statement is inaccurate, as it contradicts the goal of improving bowel regimen with fiber supplementation.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason: Ascites results from increased permeability of peritoneal capillaries, often due to inflammatory molecules like cytokines in conditions such as liver cirrhosis or portal hypertension. This allows plasma proteins and fluid to leak into the peritoneal cavity, causing fluid accumulation. This statement accurately describes the pathophysiology of ascites in liver-related disorders.
Choice B reason: Low aldosterone levels do not cause ascites; instead, high aldosterone in liver disease (e.g., cirrhosis) promotes sodium and water retention, exacerbating fluid accumulation. This statement is inaccurate, as secondary hyperaldosteronism due to reduced liver metabolism of aldosterone is a key factor in ascites development.
Choice C reason: The liver’s failure to produce clotting factors can lead to bleeding tendencies, like variceal hemorrhage, but this does not directly cause ascites. Ascites is driven by fluid leakage from capillaries, not bleeding. This statement is inaccurate, as clotting factor deficiency is unrelated to peritoneal fluid accumulation.
Choice D reason: While fluid imbalance contributes to ascites, the primary mechanism involves portal hypertension and capillary permeability, not just osmotic pressure changes. This statement is overly vague and less accurate than the specific role of inflammatory molecules increasing capillary leakage in the peritoneal cavity.
Correct Answer is C
Explanation
Choice A reason: Negative glucose in urine is normal, as the kidneys reabsorb glucose unless blood levels exceed 180 mg/dL (e.g., in diabetes). In hypertension, this finding does not indicate renal damage or require further assessment, as it aligns with normal renal function and glucose handling.
Choice B reason: Negative white blood cells in urine suggest no urinary tract infection or inflammation, a normal finding. In hypertensive patients, this does not signal kidney damage or other complications, so no additional assessment is needed, as it indicates an absence of acute inflammatory processes.
Choice C reason: Proteinuria (positive protein) indicates potential renal damage, common in hypertension due to glomerular injury from elevated pressure. It suggests impaired filtration, allowing proteins like albumin to leak into urine. This finding warrants further assessment, such as quantifying protein levels or evaluating kidney function, making it the correct choice.
Choice D reason: Creatinine in urine is normal, as it is a waste product excreted by the kidneys. While serum creatinine assesses renal function, urinary creatinine presence is expected and does not indicate pathology in hypertension, so it does not require additional assessment in this context.
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