The nurse is educating a client with Paget's disease about the importance of calcium and vitamin D intake. What is the rationale for this education?

The bone pain experienced by clients with Paget's disease is often due to microfractures and inflammation in the affected bones. The increased bone turnover and remodeling lead to weaker bones, making them more susceptible to microfractures. The inflammation at the site of these microfractures contributes to the pain, which worsens with activity.

a. Decreased blood flow to bones: Paget's disease does not typically cause decreased blood flow to bones. Instead, the condition leads to increased vascularity in affected bones.

b. Bone infarction: Bone infarction (death of bone tissue due to inadequate blood supply) is not a characteristic feature of Paget's disease. Microfractures and inflammation are more common causes of bone pain in this condition.

d. Compression of nerve roots: While nerve compression can cause bone pain, it is not a primary mechanism of pain in Paget's disease. The pain is primarily related to bone remodeling and inflammation.

Paget's disease commonly affects the axial skeleton, including the skull, spine, and pelvis. The bones in these areas are often involved in the patchy areas of increased bone density and disorganized bone remodeling seen in the condition.

a. Long bones of the extremities: While long bones can be affected by Paget's disease, the axial skeleton is more commonly involved.

c. Hands and feet: The hands and feet can be affected by Paget's disease, but the axial skeleton is more frequently involved.

d. Ribs and sternum: The ribs and sternum can be affected by Paget's disease, but the axial skeleton, including the skull, spine, and pelvis, is more commonly affected.