When talking about hypertension, we know that it can physiologically be caused by all of the following except

A. Hospital-acquired pneumonia: Pneumonia that develops 48 hours or more after hospital admission is classified as hospital-acquired pneumonia (HAP). It is caused by pathogens acquired in the hospital setting, often involving multidrug-resistant organisms such as Pseudomonas aeruginosa, Staphylococcus aureus (including MRSA), and Klebsiella pneumoniae. Patients who are intubated, have prolonged hospital stays, or have weakened immune defenses are at higher risk.

B. Immunocompromised pneumonia: Pneumonia in immunocompromised patients occurs due to weakened host defenses, such as in individuals with HIV/AIDS, those undergoing chemotherapy, or transplant recipients on immunosuppressive therapy. While these patients can develop HAP, pneumonia due to opportunistic infections like Pneumocystis jirovecii or fungal infections is categorized separately.

C. Community-acquired pneumonia: Pneumonia acquired outside the hospital or within the first 48 hours of admission is classified as community-acquired pneumonia (CAP). Typical pathogens include Streptococcus pneumoniae, Haemophilus influenzae, and Mycoplasma pneumoniae. CAP is usually less resistant to antibiotics compared to HAP.

D. Viral pneumonia: Pneumonia caused by viral pathogens such as influenza, respiratory syncytial virus (RSV), or SARS-CoV-2 is classified based on the causative agent rather than the setting in which it was acquired. Although viruses can cause both CAP and HAP, the classification of pneumonia is determined by the timing of onset and exposure risks.

A. Acute pericarditis: Acute pericarditis is characterized by inflammation of the pericardium, often presenting with pleuritic chest pain, pericardial friction rub, and diffuse ST-segment elevations on ECG. It does not typically involve pericardial thickening or chronic symptoms such as pitting edema and dyspnea on exertion.

B. Constrictive pericarditis: Chronic pericarditis with pericardial thickening suggests constrictive pericarditis, a condition where the pericardium becomes rigid and fibrotic, impairing diastolic filling and leading to heart failure symptoms such as peripheral edema, crackles in the lungs, and dyspnea on exertion. The echocardiogram findings confirm this diagnosis.

C. Pericardial effusion: Pericardial effusion refers to the accumulation of excess fluid in the pericardial sac, which can lead to cardiac tamponade if severe. However, the presence of pericardial thickening rather than fluid accumulation suggests constrictive pericarditis rather than an isolated effusion.

D. Effusion-constrictive pericarditis: This condition involves both pericardial effusion and constrictive pericarditis. While it may share some features with constrictive pericarditis, the case description primarily highlights pericardial thickening rather than significant effusion, making constrictive pericarditis the more accurate diagnosis.