(1 point). Listen.
A nurse is teaching about obesity and adipokines.
Which information should the nurse include? Obesity causes a decrease of:.
Leptin.
Adiponectin.
Homocysteine.
C-reactive protein.
The Correct Answer is A
Choice A rationale:
Leptin is a hormone produced by adipose tissue, and it plays a key role in regulating appetite and body weight. In obesity, there is an excessive amount of adipose tissue, leading to an increase in leptin levels. However, despite the higher levels of leptin, obese individuals may become resistant to its effects, leading to a decrease in the body's ability to respond to leptin signals. This condition is known as leptin resistance. Therefore, it is important to understand that obesity can cause a decrease in the effectiveness of leptin, which is critical for appetite regulation and body weight control.
Choice B rationale:
Adiponectin is another hormone produced by adipose tissue, but it has the opposite effect of leptin. Adiponectin levels are typically lower in obese individuals. While it is associated with various metabolic benefits, including improved insulin sensitivity, it doesn't directly decrease due to obesity.
Choice C rationale:
Homocysteine is not directly related to obesity. Homocysteine is an amino acid that is involved in various metabolic processes, and elevated levels are more commonly associated with cardiovascular disease and other health conditions, but not obesity.
Choice D rationale:
C-reactive protein (CRP) is a marker of inflammation in the body. While obesity is associated with chronic inflammation, it doesn't directly cause a decrease in CRP levels. In fact, obesity is more likely to lead to increased CRP levels, indicating higher levels of inflammation in the body.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A rationale:
Atherosclerosis is a complex process that involves the buildup of fatty deposits (atherosclerotic plaques) in the inner lining of arteries. The process is initiated by the oxidation of LDL (Low-Density Lipoprotein) cholesterol in the vessel wall. LDL cholesterol can become oxidized and trigger an inflammatory response in the endothelium, leading to the recruitment of immune cells and the formation of atherosclerotic lesions.
Choice B rationale:
The formation of a fatty streak in the tunica media is not the initial event that initiates atherosclerosis. Fatty streaks are a hallmark of early atherosclerosis but are a consequence of the inflammatory process triggered by the oxidation of LDL cholesterol.
Choice C rationale:
Thrombosis in the vessel lumen can be a complication of atherosclerosis but is not the primary event that initiates the atherosclerotic process.
Choice D rationale:
Endothelial injury and the release of cytokines are part of the inflammatory response that follows the initiation of atherosclerosis. However, the primary event that triggers atherosclerosis is the oxidation of LDL cholesterol in the vessel wall.
Correct Answer is A
Explanation
Choice A rationale:
High LDL (low-density lipoprotein) levels are a well-established risk factor for atherosclerosis. Elevated LDL cholesterol can lead to the deposition of cholesterol in the arterial walls, promoting the development of atherosclerotic plaques. Typically, an LDL level above 130 mg/dL is considered high.
Choice B rationale:
Low LDL levels are generally considered beneficial and are not associated with a higher risk of atherosclerosis. Lower LDL levels can be achieved through lifestyle modifications or medication and are often recommended for cardiovascular health.
Choice C rationale:
High HDL (high-density lipoprotein) levels are associated with a lower risk of atherosclerosis. HDL is considered "good" cholesterol because it helps remove excess cholesterol from the arteries. High HDL levels are typically desirable for cardiovascular health.
Choice D rationale:
Low triglyceride levels are not a primary risk factor for atherosclerosis. Elevated triglyceride levels can be associated with metabolic syndrome and other cardiovascular risk factors, but they are not as directly linked to atherosclerosis as high LDL levels.
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