A client presents to the emergency healthcare setting and diagnosed with a life-threatening ventricular dysrhythmia. Which of the following medications would the nurse begin to prepare?
Atenolol
Lisinopril
Procainamide
Adenosine
The Correct Answer is C
A. Atenolol is a beta-blocker used for various cardiac conditions but is not typically the first-line treatment for acute ventricular dysrhythmias.
B. Lisinopril is an ACE inhibitor, which is not indicated for immediate management of ventricular dysrhythmias.
C. Procainamide is an antiarrhythmic medication specifically used to treat ventricular dysrhythmias and would be appropriate in this scenario.
D. Adenosine is typically used for rapid atrial dysrhythmias, not specifically for ventricular dysrhythmias, making procainamide the better choice.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is {"A":{"answers":"C"},"B":{"answers":"A"},"C":{"answers":"B"},"D":{"answers":"D"}}
Explanation
Warfarin: Vitamin K is the primary reversal agent for anticoagulation with warfarin. It helps restore normal clotting factor production in the liver.
Heparin: Protamine sulfate is the specific reversal agent for heparin, neutralizing its anticoagulant effects.
Rivaroxaban: Andexanet alfa is a specific reversal agent for rivaroxaban and other Factor Xa inhibitors, reversing their anticoagulant effects.
Beta Blockers: While there is no specific reversal agent, glucagon can help increase heart rate and myocardial contractility in cases of beta-blocker overdose.
Correct Answer is C
Explanation
A. Calcium channel blockers primarily decrease heart rate and contractility, not increase them.
B. While they can indirectly improve oxygen supply by reducing myocardial oxygen demand, they do not directly increase systemic oxygen.
C. This option accurately describes the mechanism of action of calcium channel blockers, which decrease heart contractility and conduction through the AV node, helping to manage dysrhythmias.
D. Calcium channel blockers do not act on beta cells; they specifically inhibit calcium influx into cardiac and vascular smooth muscle cells.
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