A client tells the nurse that he has bugs in his brain and asks the nurse if the nurse can see them. Which response by the nurse is most therapeutic?
No, I don't see any bugs; you seriously can't have any bugs in your brain.
Your thinking is a little illogical; I wouldn't be able to see bugs if they were inside your brain.
You have a thought disorder and only think you have bugs in your brain; there really aren't any.
No, I don't see any bugs; that sounds scary for you.
The Correct Answer is D
Choice A reason: This response dismisses the client’s delusion, potentially increasing agitation. Schizophrenia’s mesolimbic dopamine excess drives delusional beliefs, and invalidation can heighten amygdala-driven distress, disrupting therapeutic rapport and failing to address the emotional impact of the psychotic experience.
Choice B reason: Labeling the client’s thinking as illogical is confrontational, risking escalation of paranoia. Dopamine hyperactivity in schizophrenia sustains delusions, and challenging them directly may increase amygdala activation, worsening distress and undermining trust in the therapeutic relationship.
Choice C reason: Stating the client has a thought disorder is non-therapeutic, as it dismisses the delusion without empathy. Schizophrenia’s dopamine-driven delusions require validation of feelings to reduce amygdala hyperactivity, not intellectual correction, which may alienate the client and hinder engagement.
Choice D reason: Acknowledging the delusion’s emotional impact without affirming it validates the client’s distress, reducing amygdala-driven anxiety. This therapeutic approach aligns with schizophrenia’s dopamine dysregulation, fostering trust and engagement by addressing the emotional experience of the delusion empathetically.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Choice A reason: Less assertiveness does not address anger’s neurobiology. Anger involves amygdala-driven norepinephrine and glutamate surges, and reducing assertiveness may suppress healthy expression, not resolve underlying neural hyperactivity, making this statement inaccurate for anger management.
Choice B reason: Suppressing anger is not positive, as it increases amygdala-driven stress and cortisol, potentially worsening emotional dysregulation. Healthy anger expression engages prefrontal cortex modulation, and suppression can exacerbate neural imbalances, making this statement false.
Choice C reason: Anger is a normal emotion, driven by amygdala activation and norepinephrine release. When channeled constructively, it engages prefrontal cortex regulation, facilitating conflict resolution by addressing underlying issues, making this a true and therapeutic statement about anger’s role.
Choice D reason: Punching a wall is destructive, escalating amygdala-driven aggression and norepinephrine surges without resolving conflict. It risks reinforcing maladaptive neural pathways, increasing impulsivity, and does not engage prefrontal cortex for healthy anger management, making this false.
Correct Answer is A
Explanation
Choice A reason: The amygdala regulates emotional responses like anger and aggression, with excessive norepinephrine and glutamate signaling driving hyperarousal. Its connections to the prefrontal cortex modulate impulsive behaviors, making it the primary structure for aggressive responses in emotional dysregulation.
Choice B reason: The parietal lobe processes sensory and spatial information, not emotions like anger. Aggression involves amygdala-prefrontal cortex interactions, not parietal functions, making this structure irrelevant to the neural basis of anger and aggressive behavior.
Choice C reason: The cerebellum coordinates motor activity and balance, not emotional regulation. Anger and aggression stem from amygdala-driven hyperarousal, not cerebellar function, which lacks direct involvement in emotional processing or behavioral impulsivity.
Choice D reason: The basal ganglia modulate movement and reward, not directly anger or aggression. While dopamine dysregulation may contribute, the amygdala’s role in emotional arousal is primary, making the basal ganglia secondary in this context.
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