A nurse is caring for a client who is diagnosed with a deep vein thrombosis (DVT) and is receiving heparin. The client asks the nurse how long it will take for the heparin to dissolve the clot, which of the following responses should the nurse give?
"Depending on the level of your PT/INR, the clot will begin to dissolve."
"Heparin goes not dissolve clots. It stops new clots from forming"
"It usually takes heparin at least 2 to 3 days to reach a therapeutic blood level."
"After the first dose, the clot will begin to dissolve."
The Correct Answer is B
A. PT/INR is more relevant for monitoring anticoagulant therapy like warfarin, not heparin.
B. This response is accurate; heparin prevents further clot formation but does not dissolve existing clots, which can take time for the body to reabsorb.
C. Heparin begins to exert its effects quickly, and therapeutic levels are not measured by PT/INR but by aPTT or anti-factor Xa levels.
D. Clots do not dissolve immediately after the first dose of heparin; the medication's role is to prevent clot extension.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is {"A":{"answers":"C"},"B":{"answers":"A"},"C":{"answers":"B"},"D":{"answers":"D"}}
Explanation
Warfarin: Vitamin K is the primary reversal agent for anticoagulation with warfarin. It helps restore normal clotting factor production in the liver.
Heparin: Protamine sulfate is the specific reversal agent for heparin, neutralizing its anticoagulant effects.
Rivaroxaban: Andexanet alfa is a specific reversal agent for rivaroxaban and other Factor Xa inhibitors, reversing their anticoagulant effects.
Beta Blockers: While there is no specific reversal agent, glucagon can help increase heart rate and myocardial contractility in cases of beta-blocker overdose.
Correct Answer is B
Explanation
A. Hypocalcemia is not directly linked to digoxin toxicity; rather, it is more associated with calcium channel blockers.
B. Hypokalemia increases the risk of digoxin toxicity, as low potassium levels can enhance the effects of digoxin on the heart.
C. Hyperkalemia is not directly associated with toxicity; however, it can cause complications in patients taking digoxin.
D. Hypernatremia does not have a direct impact on digoxin toxicity; monitoring potassium levels is more critical.
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