A nurse is planning care for a client who has acute glomerulonephritis. Which of the following interventions should the nurse include in the plan?
Administer antibiotics.
Encourage increased fluid intake.
Encourage frequent ambulation.
Obtain weight weekly.
The Correct Answer is A
Choice A Reason:
Administering antibiotics is a primary intervention for AGN when it is caused by a bacterial infection, such as post-streptococcal glomerulonephritis. Antibiotics help eliminate the infection and prevent further damage to the glomeruli.
Choice B Reason:
Encouraging increased fluid intake is not typically recommended for AGN, especially if the client has oliguria or edema, which are common in this condition. Fluid intake may need to be restricted to prevent fluid overload and worsening of hypertension.
Choice C Reason:
Frequent ambulation is not a priority intervention for AGN. While maintaining mobility is important, it does not directly address the renal inflammation or potential complications associated with AGN.
Choice D Reason:
Obtaining weight weekly is important for monitoring fluid status, but it is not the primary intervention. Daily weight measurements are more indicative of fluid retention or loss and are essential for closely monitoring the client's fluid balance.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A reason:
While it is true that many dietary supplements have not been tested and approved by the government, this is not the most critical information for a client taking nitroglycerin. The primary concern with dietary supplements is their potential interaction with other medications.
Choice B reason:
This is the most important statement for the nurse to include. Dietary supplements, especially those containing L-arginine or other compounds that affect blood flow, can have serious interactions with nitrates like nitroglycerin, potentially leading to dangerously low blood pressure.
Choice C reason:
The effectiveness of dietary supplements for erectile dysfunction varies, and there is indeed a lack of evidence for many of these products. However, the immediate concern for a client taking nitroglycerin is the risk of interaction, not the general effectiveness of supplements.
Choice D reason:
The cost and insurance coverage of dietary supplements can be a concern for clients, but it is not as critical as understanding the potential risks associated with their use alongside prescribed medications like nitroglycerin.
Correct Answer is B
Explanation
Choice A reason:
This choice suggests a pH of 7.50, which is indicative of alkalosis, and not typically expected in a client with chronic kidney disease (CKD). CKD often leads to metabolic acidosis due to the accumulation of acids in the body as the kidneys fail to excrete them. The HCO₃⁻ level of 20 mEq/L is slightly lower than the normal range (22-26 mEq/L), and the PaCO₂ of 32 mm Hg is within the normal range (35-45 mm Hg), but these values do not align with the expected acidosis associated with CKD.
Choice B reason:
This set of values is consistent with metabolic acidosis, which is commonly seen in clients with CKD. A pH of 7.25 indicates acidemia, and an HCO₃⁻ level of 19 mEq/L is below the normal range, suggesting a primary metabolic acidosis. The PaCO₂ of 30 mm Hg is at the lower end of the normal range, which may indicate a compensatory respiratory response to the metabolic acidosis.
Choice C reason:
A pH of 7.30, while on the lower side, is not as acidic as one would expect in a client with CKD. The HCO₃⁻ level of 26 mEq/L is within the normal range, and a PaCO₂ of 50 mm Hg suggests respiratory acidosis, which is not the primary disorder in CKD.
Choice D reason:
This choice indicates a pH of 7.55, which is too alkaline and not characteristic of CKD, where metabolic acidosis is the expected finding. An HCO₃⁻ level of 30 mEq/L is higher than the normal range, suggesting metabolic alkalosis. The PaCO₂ of 31 mm Hg is slightly below the normal range, possibly indicating a compensatory response, but it does not align with the metabolic acidosis typically seen in CKD.
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