A nurse is reviewing the laboratory results for a male adult client who is at risk for peripheral arterial disease due to atherosclerosis. The nurse should identify which of the following results places the client at risk :
Triglycerides : 130 mg/dL
HDL : 84 mg/dL
Blood glucose : 92 mg/dL
LDL : 172 mg/dL
The Correct Answer is D
Choice A reason : The triglyceride level of 130 mg/dL is within the normal range, which is less than 150 mg/dL for adults. Therefore, this result does not place the client at risk for peripheral arterial disease.
Choice B reason : An HDL level of 84 mg/dL is considered good as it is higher than the minimum recommended level of 40 mg/dL for males and 50 mg/dL for females. Higher levels of HDL are protective against heart disease.
Choice C reason : A blood glucose level of 92 mg/dL is within the normal fasting range of 70 to 99 mg/dL. This indicates that the client does not have diabetes, which is a risk factor for peripheral arterial disease.
Choice D reason : An LDL level of 172 mg/dL is considered high since the optimal level for LDL is less than 100 mg/dL. High levels of LDL cholesterol can lead to atherosclerosis, increasing the risk for peripheral arterial disease.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason : Aspirin is well-known for its antiplatelet properties, which inhibit platelet aggregation and thus prevent the formation of new blood clots. This is particularly important following a myocardial infarction, as it helps to prevent further clotting events that could lead to additional heart attacks or strokes. Aspirin's effect on platelets is so significant that it is often one of the first medications administered in the setting of acute coronary syndrome.
Choice B reason : While aspirin does have analgesic properties, this is not the primary reason it is prescribed post-myocardial infarction. The analgesic effect of aspirin is more commonly utilized for minor pains and aches, and it is not sufficient for the pain associated with coronary artery disease.
Choice C reason : Aspirin can reduce fever, but this is not relevant to its use in coronary artery disease. Fever reduction is not a concern when prescribing aspirin for myocardial infarction patients, as the primary goal is to manage the risk of thrombosis.
Choice D reason : Aspirin does have anti-inflammatory effects, but again, this is not the primary reason for its prescription following a myocardial infarction. While inflammation plays a role in atherosclerosis and coronary artery disease, the anti-inflammatory properties of aspirin are not the main focus in the context of post-myocardial infarction treatment.
Correct Answer is B
Explanation
Choice A reason : Epinephrine is an adrenergic agonist primarily used in the management of cardiac arrest, anaphylaxis, and severe asthma attacks. It is not used to reverse the effects of warfarin overdose. Warfarin acts as an anticoagulant by inhibiting vitamin K-dependent clotting factors, and epinephrine has no role in this mechanism.
Choice B reason : Vitamin K is the antidote for warfarin toxicity. Warfarin works by inhibiting the vitamin K-dependent clotting factors II, VII, IX, and X. In the event of an overdose, vitamin K is administered to reverse the anticoagulant effects of warfarin and restore the clotting factor levels to normal. The administration can be oral or intravenous, depending on the severity of the overdose and the urgency of the situation.
Choice C reason : Atropine is an anticholinergic drug used to treat bradycardia (slow heart rate) and as an antidote for organophosphate poisoning. It does not have a role in reversing warfarin overdose as it does not affect the clotting cascade or vitamin K metabolism.
Choice D reason : Protamine is used to reverse the effects of heparin, another anticoagulant, but not warfarin. Protamine sulfate binds to heparin, forming a stable complex and neutralizing its anticoagulant effects. Since warfarin's mechanism of action is different from heparin's, protamine is not effective in reversing warfarin toxicity.
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