Bile salts break up the fat globule into smaller fat droplets. This role of bile salts is best described as
lipid absorption
lipid emulsification
lipid ingestion
lipid digestion
The Correct Answer is B
A. Lipid absorption: Absorption occurs in the small intestine after fats have been digested into smaller molecules. Bile salts do not absorb lipids directly.
B. Lipid emulsification: Bile salts break large fat globules into smaller droplets in a process called emulsification, which increases the surface area for digestive enzymes like lipase to act.
C. Lipid ingestion: Ingestion refers to eating, not the chemical processing of fats.
D. Lipid digestion: Digestion is primarily performed by lipase, not bile salts. Bile salts prepare fats for digestion.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A. Involves hypothalamic osmoreceptor detection of ion concentration: Osmoreceptors detect osmolarity (solute concentration), not specifically sodium regulation.
B. Is due to specific sodium receptors in the hypothalamus: There are no specific sodium receptors; sodium regulation is largely hormonal.
C. Is linked to blood pressure: Sodium levels affect blood volume and pressure through water retention. The renin-angiotensin-aldosterone system (RAAS) helps regulate both sodium and blood pressure.
D. Involves aldosterone, a hormone that increases sodium excretion in the kidneys: Aldosterone increases sodium reabsorption, not excretion.
Correct Answer is B
Explanation
A. Lipolysis, glycogenolysis, beta oxidation: These refer to fat and glycogen metabolism, not the complete oxidation of glucose.
B. Glycolysis, citric acid (Krebs) cycle, electron transport chain, oxidative phosphorylation: This is the correct and complete pathway for glucose metabolism leading to ATP production.
C. Glycogenesis, lipogenesis, electron transport chain: Glycogenesis and lipogenesis are anabolic processes, not catabolic.
D. Gluconeogenesis, citric acid (Krebs) cycle, lipolysis: These are mixed pathways that do not directly represent glucose oxidation.
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