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Harry Eastlack died from a condition where his connective tissues and muscles turned to bone. What did he die from?

A.

osteogenesis imperfecta

B.

osteopetrosis

C.

chondroidal ossification

D.

fibrodysplasia ossificans progressiva

E.

osteoarthritis

Answer and Explanation

The Correct Answer is D

A. osteogenesis imperfecta
This is brittle bone disease, caused by defective collagen, not muscle turning into bone.

B. osteopetrosis
This is a rare condition involving overly dense bones due to osteoclast failure, not soft tissue ossification.

C. chondroidal ossification
This is not a recognized clinical condition. Ossification of cartilage does occur in development but doesn’t describe Harry's case.

D. fibrodysplasia ossificans progressiva (FOP)
FOP is a rare genetic disorder where soft connective tissues such as muscles, tendons, and ligaments turn into bone, progressively restricting movement and ultimately leading to complications that may be fatal.

E. osteoarthritis
This involves cartilage degeneration at joints due to wear and tear-not ectopic bone formation in soft tissues.


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Related Questions

Correct Answer is C

Explanation

A. he could develop brittle bones
Brittle bones are more associated with osteoporosis or osteogenesis imperfecta, not primarily from vitamin D deficiency due to lack of sunlight.

B. he could develop very dense bones
Dense bones are seen in conditions like osteopetrosis, which results from defective osteoclast activity-not lack of sunlight.

C. he could develop soft, bowed bones
Lack of sunlight leads to vitamin D deficiency, impairing calcium absorption, which causes rickets in children and osteomalacia in adults, both of which involve soft, weakened bones that may bow under body weight.

D. he could develop nightmares
While isolation might impact mental health, this has no direct link to skeletal effects or bone physiology.

Correct Answer is D

Explanation

A. parathyroid hormone is released, decreasing osteoclast activity
Parathyroid hormone (PTH) is released when calcium levels are low, not high. Also, PTH increases, not decreases, osteoclast activity to raise serum calcium.

B. calcitonin hormone is released, increasing osteoclast activity
Calcitonin is released during high calcium levels, but it works to decrease osteoclast activity, not increase it.

C. parathyroid hormone is released, increasing osteoclast activity
This would occur in response to low calcium, which is not the case here. Mary has excess calcium.

D. calcitonin hormone is released, decreasing osteoclast activity
With elevated calcium levels, the thyroid gland releases calcitonin, which works to inhibit osteoclasts to reduce calcium release from bone, helping to restore balance.

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