What have you learned about ventilation?
Hypoventilation causes hypocapnia
Hypoventilation causes alkalosis
Hyperventilation causes acidosis
Hyperventilation causes hypocapnia
The Correct Answer is D
A. Hypoventilation causes hypocapnia: Hypoventilation leads to inadequate removal of carbon dioxide (CO₂), resulting in hypercapnia rather than hypocapnia. This occurs because decreased ventilation reduces gas exchange efficiency, allowing CO₂ to accumulate in the blood, which can cause respiratory acidosis.
B. Hypoventilation causes alkalosis: Hypoventilation leads to respiratory acidosis rather than alkalosis. When ventilation is insufficient, CO₂ builds up in the bloodstream, leading to an increase in hydrogen ion concentration and a subsequent drop in blood pH. This is commonly seen in conditions like chronic obstructive pulmonary disease (COPD), neuromuscular disorders, and opioid overdose.
C. Hyperventilation causes acidosis: Hyperventilation results in excessive elimination of CO₂, leading to a decrease in hydrogen ion concentration and an increase in blood pH, causing respiratory alkalosis rather than acidosis. Acidosis occurs when CO₂ retention leads to an increase in hydrogen ion concentration, which is the opposite of what happens with hyperventilation.
D. Hyperventilation causes hypocapnia: Hyperventilation leads to an excessive exhalation of CO₂, causing a reduction in blood CO₂ levels (hypocapnia). This can result in respiratory alkalosis, leading to symptoms such as dizziness, lightheadedness, tingling sensations, and even syncope. It is often seen in conditions like anxiety, panic attacks, fever, or high-altitude exposure.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A. Bradycardia: Bradycardia, which is a slower than normal heart rate, is not a typical clinical manifestation of pleural effusion with empyema. In fact, fever and infection usually lead to tachycardia (an increased heart rate) as the body attempts to respond to infection.
B. Pleural pain: Pleural pain is a common symptom associated with pleural effusion and empyema, resulting from irritation of the pleura due to inflammation or infection.
C. Cough: A cough can occur with pleural effusion and empyema, often as a response to irritation or pressure in the thoracic cavity. It may also be associated with underlying lung conditions contributing to the effusion.
D. Fever: Fever is a typical clinical manifestation of empyema, indicating an infectious process. The presence of infection in the pleural space often results in systemic signs of infection, such as fever.
Correct Answer is A
Explanation
A. Injury to the endothelial cells that line the artery walls: The development of atherosclerosis begins with damage to the endothelial cells of the arterial wall. This injury can be caused by various factors, including hypertension, smoking, high cholesterol, and diabetes. Once the endothelium is injured, it becomes more permeable, allowing lipids and inflammatory cells to penetrate and accumulate, leading to the formation of atherosclerotic plaques.
B. Release of the platelet-derived growth factor: While platelet-derived growth factor (PDGF) plays a role in the proliferation of smooth muscle cells and the progression of atherosclerosis, it is not the initiating event. PDGF is released in response to endothelial injury and inflammation but does not cause the initial damage itself.
C. Macrophages adhere to vessel walls: The adherence of macrophages to the vessel walls occurs after the initial endothelial injury. Once the endothelium is damaged, macrophages migrate to the site and contribute to the inflammatory response and plaque formation, but this is not the initiating event.
D. Release of inflammatory cytokines: Inflammatory cytokines are part of the response that follows endothelial injury and play a role in the progression of atherosclerosis. However, the release of these cytokines is a consequence of the initial injury rather than the initiating event.
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