The nurse caring for a woman hospitalized for hyperemesis gravidarum would expect that the initial treatment would involve:
Enteral nutrition (TPN) to correct nutritional deficits
Small frequent meals
Corticosteroids to reduce inflammation
IV therapy to correct fluid and electrolyte imbalances
The Correct Answer is D
A. Enteral nutrition (TPN) to correct nutritional deficits. Total parenteral nutrition (TPN) is only considered in severe cases where oral and IV hydration fail. The initial treatment focuses on correcting dehydration and electrolyte imbalances before considering more invasive nutritional support.
B. Small frequent meals. While small, frequent meals may help manage nausea in mild cases of pregnancy-related nausea and vomiting, they are not sufficient for treating hyperemesis gravidarum, which involves severe, persistent vomiting leading to dehydration and electrolyte imbalances.
C. Corticosteroids to reduce inflammation. Corticosteroids are not the first-line treatment for hyperemesis gravidarum. They may be used in refractory cases where standard treatments fail, but IV fluids and antiemetics are prioritized initially.
D. IV therapy to correct fluid and electrolyte imbalances. The primary concern in hyperemesis gravidarum is severe dehydration and electrolyte disturbances due to excessive vomiting. IV fluids, often with electrolytes and thiamine, are the first step in stabilizing the patient before introducing oral intake.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A. Jaundice. Oxytocin does not cause jaundice in the mother. Jaundice is typically related to liver dysfunction, hemolysis, or bile obstruction and is not an expected side effect of oxytocin administration.
B. Dehydration. While prolonged labor induction may contribute to dehydration if fluid intake is insufficient, oxytocin itself does not directly cause dehydration. IV fluids are typically administered alongside oxytocin to maintain hydration during labor.
C. Uterine hyperstimulation. Uterine hyperstimulation (tachysystole), defined as more than five contractions in 10 minutes, is a serious complication of oxytocin administration. It can lead to fetal distress, uterine rupture, and impaired placental perfusion. If hyperstimulation occurs, the nurse should discontinue oxytocin, reposition the patient, provide oxygen, and notify the provider.
D. Maternal bradycardia. Oxytocin does not typically cause bradycardia. Maternal side effects are more commonly tachycardia, hypertension, or fluid retention. Fetal bradycardia, however, can occur if uterine hyperstimulation leads to fetal hypoxia.
Correct Answer is D
Explanation
A. Maintaining euglycemia in labor reduces the need for insulin postpartum. While insulin requirements typically decrease after delivery due to the loss of placental hormones that cause insulin resistance, the primary reason for tight glucose control during labor is to prevent neonatal complications rather than reducing postpartum insulin needs.
B. A blood glucose level above 110 puts the client at risk for infection in labor. Poorly controlled diabetes can increase infection risk over time, but transient hyperglycemia in labor is not a direct cause of infection. The focus of glucose management during labor is to prevent neonatal hypoglycemia rather than maternal infection.
C. More insulin will be available for fetal use via placental transfer. Insulin does not cross the placenta, so maternal insulin therapy does not provide insulin to the fetus. However, maternal hyperglycemia leads to increased fetal insulin production, which can cause neonatal hypoglycemia after birth.
D. An elevated blood glucose in labor increases the risk of neonatal hypoglycemia. Maternal hyperglycemia causes the fetus to produce excessive insulin in utero. After birth, when the maternal glucose supply is suddenly cut off, the infant’s high insulin levels can cause a rapid drop in blood glucose, leading to neonatal hypoglycemia, which can be dangerous if not managed properly.
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