The nurse is caring for a client with infectious endocarditis. Which information obtained from the health history could have contributed to the development of the disease progress?
Hypertension treated with medications
Prostate surgery six months prior
Use of intravenous substances
Stroke diagnosed one year ago
The Correct Answer is C
A) Hypertension treated with medications
Hypertension (high blood pressure) itself is not a direct cause of infectious endocarditis. While untreated or poorly managed hypertension can contribute to cardiovascular complications, it is not typically a risk factor for developing infectious endocarditis. Infectious endocarditis is more commonly associated with conditions that directly involve the heart valves or blood stream, such as intravenous drug use, prior heart valve disease, or certain invasive procedures.
B) Prostate surgery six months prior
While certain surgeries, such as dental or urinary tract procedures, can increase the risk of infectious endocarditis due to transient bacteremia, prostate surgery by itself is not a major risk factor for this condition. Unless there was a complication during the surgery that resulted in bacteremia (e.g., infection), B is not the most likely contributor to the development of infectious endocarditis.
C) Use of intravenous substances
The use of intravenous (IV) substances, especially illicit drugs, is a major risk factor for the development of infectious endocarditis. Intravenous drug use, particularly when non-sterile needles or contaminated substances are used, can introduce bacteria directly into the bloodstream, leading to bacteremia.
D) Stroke diagnosed one year ago
While a history of stroke may indicate underlying cardiovascular disease or embolic events, it is not directly related to the development of infectious endocarditis. Stroke can occur as a complication of infectious endocarditis, particularly if emboli from infected valves travel to the brain. However, a prior stroke itself does not directly contribute to the development of endocarditis.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A) Ensure bed alarm is on when leaving patient's room:
The cerebellum plays a critical role in coordinating movement, balance, and motor control. An injury to this area can lead to impaired coordination and dysfunction in balance, making it difficult for the patient to perform the Romberg test (which evaluates balance and proprioception). Given that the patient cannot complete the Romberg test, this suggests the risk of falling or injuring themselves due to impaired balance and coordination. The priority nursing intervention is to ensure safety by using a bed alarm to alert staff if the patient attempts to get out of bed, thereby preventing falls.
B) Vary schedule to prevent memorization and boredom:
While changing the patient's routine may help with engagement, it is not the priority intervention for a patient with cerebellar injury. The primary concern in this situation is safety due to the patient's impaired balance. Addressing issues related to cognitive function or boredom may be important but comes after ensuring the patient’s physical safety.
C) Clarify misinformation and reorient when confused:
Reorientation may be necessary if the patient is confused or disoriented, but this is not the primary concern related to cerebellar injury. The patient’s balance and motor coordination are the most pressing issues. While mental clarity is important, preventing falls due to impaired motor control is the immediate priority.
D) Deep breathing and incentive spirometer education:
Deep breathing exercises and using an incentive spirometer are essential to prevent respiratory complications, particularly after surgery or immobility. However, in this case, the priority concern is the patient's safety related to impaired balance from the cerebellar injury. Respiratory interventions are important but should be addressed after ensuring that the patient is safe from falls and other immediate physical risks.
Correct Answer is ["A","B","D"]
Explanation
A) Tachycardia: Tachycardia is commonly seen in left-sided heart failure as the heart attempts to compensate for the reduced cardiac output. To maintain adequate perfusion, the body increases the heart rate. The sympathetic nervous system is activated, causing an increase in heart rate to try to pump blood more efficiently despite the reduced pumping ability of the left ventricle.
B) Crackles: Crackles (also known as rales) are typically heard upon auscultation of the lungs in patients with left-sided heart failure. When the left ventricle fails to effectively pump blood, it causes blood to back up into the lungs, resulting in pulmonary congestion. This leads to fluid accumulation in the alveoli, causing crackling sounds during inhalation.
C) Ascites: Ascites is more commonly seen in right-sided heart failure or in cases of congestive heart failure where both sides of the heart are affected. Ascites is the accumulation of fluid in the abdomen, which is a consequence of right-sided heart failure causing blood to back up into the abdomen. In left-sided heart failure, ascites is generally not a primary symptom unless the failure becomes severe and involves both sides of the heart.
D) Dyspnea: Dyspnea, or shortness of breath, is a hallmark symptom of left-sided heart failure. The left ventricle's inability to pump blood efficiently leads to pulmonary congestion, which causes fluid to accumulate in the lungs. This fluid buildup reduces the lung's ability to exchange oxygen, resulting in difficulty breathing, especially on exertion or when lying down (orthopnea).
E) Peripheral edema: Peripheral edema is more characteristic of right-sided heart failure due to the backup of blood in the systemic circulation. While it can occur in cases of biventricular heart failure (both right and left sides of the heart are affected), it is not the primary finding in left-sided heart failure. Left-sided heart failure typically presents with pulmonary symptoms rather than systemic symptoms like peripheral edema.
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