Which clinical manifestations would you assess for in a painter with pulmonary hypertension?
(Select All that Apply.)
Jugular vein distention
Peripheral edema
Ronchi bilaterally
Dyspnea on exertion
Systemic blood pressure greater than 130/90 mm HG
Correct Answer : A,B,D
A. Jugular vein distention: Pulmonary hypertension increases right ventricular afterload, leading to right heart failure. As the right ventricle struggles to pump against elevated pulmonary pressures, blood backs up into the systemic circulation, causing jugular vein distention (JVD).
B. Peripheral edema: Right-sided heart failure due to pulmonary hypertension leads to venous congestion and fluid retention, resulting in peripheral edema. Swelling is commonly observed in the lower extremities due to increased venous hydrostatic pressure.
C. Ronchi bilaterally: Rhonchi are low-pitched breath sounds typically associated with airway obstruction due to secretions, commonly seen in conditions like chronic bronchitis or pneumonia. Pulmonary hypertension primarily affects the pulmonary vasculature rather than the airways, making rhonchi an unlikely manifestation.
D. Dyspnea on exertion: Pulmonary hypertension increases pressure in the pulmonary arteries, reducing oxygen exchange and causing exertional dyspnea. This symptom is often one of the earliest clinical manifestations as increased pulmonary vascular resistance impairs oxygen delivery.
E. Systemic blood pressure greater than 130/90 mm Hg: Pulmonary hypertension specifically affects the pulmonary circulation, not systemic arterial pressure. While pulmonary hypertension can eventually contribute to left heart dysfunction, it does not directly cause systemic hypertension.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
A. By causing dysrhythmias as a result of hyperkalemia: Angiotensin II does not directly cause dysrhythmias through hyperkalemia. While hyperkalemia can lead to dysrhythmias, angiotensin II primarily acts as a vasoconstrictor and does not directly influence potassium levels.
B. By increasing peripheral vasoconstriction: Angiotensin II increases peripheral vascular resistance through vasoconstriction, which raises blood pressure. This increased resistance requires the heart to work harder to pump blood, thereby increasing the workload on the heart following a myocardial infarction.
C. By stimulating the sympathetic nervous system: Angiotensin II does stimulate the sympathetic nervous system, which can increase heart rate and contractility; however, the direct effect on increasing workload is more prominently due to peripheral vasoconstriction.
D. By reducing the contractility of the myocardium: Angiotensin II does not reduce contractility; in fact, it can enhance contractility in some contexts. It primarily increases the workload of the heart through vasoconstriction and increased afterload rather than reducing contractility.
Correct Answer is D
Explanation
A. Vernelli's triad; Venous stasis; Hypercoagulable states; Venous intimal damage: Vernelli's triad is not a recognized term in thrombus formation. Venous stasis, hypercoagulable states, and venous intimal damage are relevant factors, but the correct terminology is Virchow's triad.
B. Vernelli's triad: Hyperlipidemia; hypercoagulable states, venous stasis: This option inaccurately references Vernelli's triad and includes hyperlipidemia, which is not one of the classic factors associated with thrombus formation. The correct factors should be named according to Virchow's triad.
C. Virchow's triad; Hyperlipidemia, Hypercoagulable states, positive D-dimer: Although Virchow's triad is correctly identified, hyperlipidemia and positive D-dimer are not part of the classic factors involved in thrombus formation. The classic factors are venous stasis, hypercoagulable states, and venous intimal damage.
D. Virchow triad; Venous stasis; Venous intimal damage; Hypercoagulable states: This option accurately identifies Virchow's triad and lists the three key factors involved in thrombus formation: venous stasis, venous intimal damage, and hypercoagulable states.
Whether you are a student looking to ace your exams or a practicing nurse seeking to enhance your expertise , our nursing education contents will empower you with the confidence and competence to make a difference in the lives of patients and become a respected leader in the healthcare field.
Visit Naxlex, invest in your future and unlock endless possibilities with our unparalleled nursing education contents today
Report Wrong Answer on the Current Question
Do you disagree with the answer? If yes, what is your expected answer? Explain.
Kindly be descriptive with the issue you are facing.