Which electrolyte imbalance would the nurse expect to find in a client receiving acetazolamide?
Metabolic acidosis
Metabolic alkalosis
Hyperbicarbontemia
Hypercalcemia
The Correct Answer is A
A. Metabolic acidosis: Acetazolamide is a carbonic anhydrase inhibitor that promotes the excretion of bicarbonate, leading to a decrease in bicarbonate levels and resulting in metabolic acidosis.
B. Metabolic alkalosis: Acetazolamide causes a decrease in bicarbonate, which would lead to acidosis, not alkalosis.
C. Hyperbicarbonatemia: Acetazolamide reduces bicarbonate reabsorption, leading to a decrease in bicarbonate levels, not an increase.
D. Hypercalcemia: Acetazolamide does not cause hypercalcemia; it affects acid-base balance and bicarbonate levels.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A. Hypotension: Hypotension is not a common interaction between oxybutynin and haloperidol.
B. Lowered plasma concentrations: There is no significant interaction that would lead to lowered plasma concentrations of either medication.
C. Increased effect of the antispasmodic drug: Both oxybutynin and haloperidol have anticholinergic properties. When combined, these properties can increase the risk of anticholinergic side effects, such as dry mouth, constipation, and urinary retention, enhancing the effect of oxybutynin.
D. Decreased effect of the antipsychotic drug: There is no evidence that oxybutynin would decrease the effect of haloperidol.
Correct Answer is C
Explanation
A. Dopamine receptors: Dopamine receptors are involved in the regulation of movement and mood but are not the primary receptors affected by oxybutynin.
B. Beta adrenergic receptors: Beta adrenergic receptors are involved in smooth muscle relaxation, but oxybutynin targets the muscarinic receptors.
C. Muscarinic receptors: Oxybutynin works by blocking muscarinic receptors in the bladder, thereby reducing overactive bladder symptoms like urgency and frequency.
D. Alpha adrenergic receptors: These receptors are involved in vasoconstriction and are not the primary target of oxybutynin.
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