Which of the following situations stimulates the skin to produce Vitamin D?
Presence of calcium
Exposure to ultraviolet light
Exposure to cold temperature
Presence of parathyroid hormone
The Correct Answer is B
a. Presence of calcium: Calcium does not directly stimulate the production of Vitamin D in the skin.
b. Exposure to ultraviolet light: UVB rays from sunlight stimulate the skin to produce Vitamin D3 (cholecalciferol).
c. Exposure to cold temperature: Cold temperature does not affect Vitamin D production.
d. Presence of parathyroid hormone: Parathyroid hormone helps regulate calcium levels and can stimulate the conversion of Vitamin D to its active form in the kidneys, but it does not stimulate its production in the skin.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
a. Sodium: Correct. Sodium (Na⁺) is the primary extracellular cation and plays a crucial role in maintaining osmotic pressure and fluid balance in the body.
b. Magnesium: Magnesium (Mg²⁺) is important for many cellular functions but is not a major contributor to osmotic pressure compared to sodium.
c. Calcium: Calcium (Ca²⁺) is vital for bone health and cellular processes but does not primarily influence osmotic pressure.
d. Potassium: Potassium (K⁺) is the main intracellular cation and affects cell function, but sodium has a more significant role in osmotic pressure regulation.
Correct Answer is D
Explanation
a. Decrease in hydrogen ions: A decrease in hydrogen ions (H+) would indicate a condition of alkalosis, not acidosis.
b. Decrease in carbon dioxide: A decrease in carbon dioxide (CO2) usually leads to respiratory alkalosis because CO2 is acidic in nature.
c. Decrease in ketones: Ketones are acidic compounds, and their decrease is not associated with acidosis. In fact, an increase in ketones can cause ketoacidosis.
d. Decrease in bicarbonate ions: Bicarbonate (HCO3-) is a base, and a decrease in its concentration can lead to metabolic acidosis, as it reduces the buffering capacity of the blood against excess H+.
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