A nurse is assessing a patient diagnosed with Addison's disease. Which clinical manifestations are expected?

A. Diminished serum albumin levels cause water to shift from blood to tissue: In cirrhosis, liver dysfunction leads to decreased production of albumin, a protein that helps maintain oncotic pressure. Low albumin levels cause fluid to shift from the vascular space into the tissues, resulting in ascites and peripheral edema.

B. Portal hypotension causes a fluid shift from the abdominal cavity into the portal veins: Portal hypertension, not hypotension, is a common feature of cirrhosis, but it leads to ascites by increasing pressure in the portal venous system, not by shifting fluid into the portal veins.

C. Hypoaldosteronism causes a fluid volume deficit: shifting water from blood into tissue: Cirrhosis often leads to hyperaldosteronism, not hypoaldosteronism, resulting in sodium and water retention, which contributes to edema.

D. Aberrations of the portal system cause a back-up of blood that leads to hydronephrosis: Hydronephrosis is related to obstruction of the urinary tract, not a complication of portal hypertension or cirrhosis.

A. Non-bloody diarrhea with abdominal cramps: These symptoms are not typical of pancreatitis. They are more common in gastrointestinal infections or irritable bowel syndrome.

B. Belching and burping within one hour after eating: These symptoms are usually associated with gastroesophageal reflux disease (GERD) or dyspepsia, not pancreatitis.

C. Severe epigastric pain that radiates to his back: Severe epigastric pain that radiates to the back is a hallmark symptom of acute pancreatitis.

D. Heartburn that worsens when lying down: This symptom is more characteristic of GERD rather than pancreatitis.