ADH release from the posterior pituitary is stimulated by which process?
Low blood pressure sensed by baroreceptors in the kidneys.
Low osmolality sensed by osmoreceptors in the kidneys.
High plasma osmolarity sensed by osmoreceptors in the hypothalamus.
High concentration of potassium sensed by chemoreceptors in the carotid body.
The Correct Answer is C
Choice A rationale: Low blood pressure sensed by baroreceptors in the kidneys would stimulate the renin-angiotensin-aldosterone system, which regulates blood pressure and sodium balance.
Choice B rationale: Low osmolality sensed by osmoreceptors in the kidneys would
indicate that the body has excess water and needs to excrete it, which would inhibit ADH release.
Choice C rationale: ADH, or antidiuretic hormone, is a peptide hormone that regulates the water balance in the body. It is released from the posterior pituitary gland in response to signals from the hypothalamus. When the plasma osmolarity, or the concentration of solutes in the blood, is high, it means that the body is dehydrated and needs to conserve water. The osmoreceptors in the hypothalamus detect this change and stimulate the release of ADH, which acts on the kidneys to increase water
reabsorption and decrease urine output. This helps to lower the plasma osmolarity and restore the water balance.
Choice D rationale: High concentration of potassium sensed by chemoreceptors in the carotid body would affect the acid-base balance and respiratory rate, but not ADH
release.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A rationale: These side effects are not related to spironolactone use.
Choice B rationale: These are common side effects of spironolactone, as it causes potassium retention and sodium loss.
Choice C rationale: These side effects are not related to spironolactone use.
Choice D rationale: These side effects are not related to spironolactone use.
Correct Answer is D
Explanation
Choice A rationale: Phenelzine is an antidepressant that typically does not affect blood glucose levels significantly.
Choice B rationale: Allopurinol, used for gout, is not known to significantly impact blood glucose levels.
Choice C rationale: Metoprolol, a beta-blocker, might mask some symptoms of hypoglycemia but is not typically associated with causing hyperglycemia.
Choice D rationale: Methylprednisolone, a corticosteroid, can elevate blood glucose levels and might contribute to hyperglycemia in a person with diabetes mellitus.
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