Appropriate therapy for prerenal kidney injury includes:
Protein restriction.
Potassium supplementation.
Fluid administration to increase cardiac output.
Fluid restriction.
The Correct Answer is C
Choice A reason: Protein restriction is not a primary treatment for prerenal kidney injury. While managing protein intake can be important in chronic kidney disease to reduce the workload on the kidneys, it is not directly relevant to the acute management of prerenal kidney injury, which primarily involves restoring adequate blood flow to the kidneys.
Choice B reason: Potassium supplementation is not typically necessary for prerenal kidney injury. In fact, in many cases of kidney injury, there is a risk of hyperkalemia (elevated potassium levels) due to reduced kidney function. Therefore, potassium levels need to be monitored, but supplementation is not a standard treatment.
Choice C reason: Fluid administration to increase cardiac output is the appropriate therapy for prerenal kidney injury. Prerenal kidney injury is caused by reduced blood flow to the kidneys, often due to factors like hypovolemia (low blood volume) or heart failure. Administering fluids helps to restore adequate blood flow and perfusion to the kidneys, which can improve kidney function and resolve the prerenal injury.
Choice D reason: Fluid restriction is not appropriate for prerenal kidney injury, which is characterized by reduced blood flow to the kidneys. In cases where hypovolemia or low cardiac output is the cause, restricting fluids would worsen the condition. Instead, increasing fluid intake helps to restore adequate perfusion to the kidneys.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Choice A reason: Chronic obstructive pulmonary disease (COPD) is a progressive lung disease characterized by airflow obstruction and is typically caused by long-term exposure to irritants such as cigarette smoke. While COPD can lead to pulmonary hypertension as a complication, it is not caused by chronic pulmonary hypertension. Therefore, chronic pulmonary hypertension does not directly cause COPD.
Choice B reason: Respiratory acidosis occurs when the lungs cannot remove enough carbon dioxide from the body, leading to a decrease in blood pH. While it can be associated with various respiratory conditions, it is not a direct complication of chronic pulmonary hypertension. Chronic pulmonary hypertension primarily affects the pressure within the pulmonary arteries and the right side of the heart rather than directly causing respiratory acidosis.
Choice C reason: Pulmonary emboli are blood clots that travel to the lungs and can cause sudden obstruction of the pulmonary arteries. While pulmonary emboli can lead to acute pulmonary hypertension, they are not typically caused by chronic pulmonary hypertension. Chronic pulmonary hypertension is usually a result of long-term conditions affecting the pulmonary vasculature, whereas pulmonary emboli are acute events.
Choice D reason: Right heart failure, also known as Cor Pulmonale, is a direct complication of chronic pulmonary hypertension. Chronic pulmonary hypertension increases the pressure in the pulmonary arteries, causing the right side of the heart to work harder to pump blood through the lungs. Over time, this increased workload can lead to right ventricular hypertrophy (thickening of the heart muscle) and eventual failure. Cor Pulmonale is a significant consequence of chronic pulmonary hypertension and can lead to symptoms such as edema, ascites, and fatigue.
Correct Answer is C
Explanation
Choice A reason: Compensated metabolic acidosis is characterized by a low pH (acidemia) with a compensatory decrease in PaCO2 due to hyperventilation, and typically a low HCO3 as well. In this scenario, the pH is elevated (alkalemia), which rules out metabolic acidosis.
Choice B reason: Compensated metabolic alkalosis is characterized by a high pH (alkalemia) with a compensatory increase in PaCO2 due to hypoventilation, and a high HCO3. In this case, the PaCO2 is low rather than high, and the HCO3 is within the normal range, which rules out metabolic alkalosis.
Choice C reason: Respiratory alkalosis is characterized by a high pH (alkalemia) and a low PaCO2, indicating that the alkalosis is due to excessive loss of CO2 through hyperventilation. The HCO3 is usually normal or slightly decreased as a compensatory mechanism. The provided ABG values (pH = 7.53, PaCO2 = 23, HCO3 = 22) are consistent with respiratory alkalosis, where the elevated pH and low PaCO2 point towards hyperventilation as the cause of the alkalemia.
Choice D reason: Uncompensated respiratory alkalosis would present with a high pH and low PaCO2, without significant compensation by the kidneys (i.e., HCO3 would still be normal). In this scenario, the ABG values fit the description of respiratory alkalosis, but it is considered compensated since the HCO3 is within the normal range, indicating some level of renal compensation.
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