The nurse administers furosemide IV to a patient with acute pulmonary edema. Which action is most important during administration?
Restrict the patient's fluid intake.
Ensure the patient voids before administration.
Administer the dose rapidly to achieve quick results.
Monitor the patient for tinnitus.
The Correct Answer is D
(A) Restrict the patient’s fluid intake: Fluid restrictions are important but not the priority during administration. Monitoring for adverse effects like ototoxicity is more critical.
(B) Ensure the patient voids before administration: Furosemide is a diuretic, but voiding before administration is not required.
(C) Administer the dose rapidly to achieve quick results: Rapid IV push can cause ototoxicity and hypotension. It should be given slowly over 1-2 minutes.
(D) Monitor the patient for tinnitus: High-dose or rapid IV administration of furosemide can cause ototoxicity, leading to tinnitus and hearing loss.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is {"A":{"answers":"A"},"B":{"answers":"A"},"C":{"answers":"B"},"D":{"answers":"A"},"E":{"answers":"B"},"F":{"answers":"C"},"G":{"answers":"C"}}
Explanation
|
Prerenal |
Intrarenal |
Postrenal |
|
Severe Dehydration Sepsis CHF (Congestive Heart Failure) |
Acute Tubular Necrosis (ATN) Gentamicin (Aminoglycoside antibiotic toxicity) |
Benign Prostatic Hyperplasia (BPH) Renal Calculi (Kidney Stones) |
Severe Dehydration: Prerenal
Decreased blood flow to the kidneys due to low volume.
Sepsis: Prerenal
Hypotension from systemic infection reduces kidney perfusion.
CHF (Congestive Heart Failure): Prerenal
Reduced cardiac output leads to inadequate renal perfusion.
Benign Prostatic Hyperplasia (BPH): Postrenal
Urinary outflow obstruction causes back pressure on the kidneys
Renal Calculi (Kidney Stones): Postrenal
Blockage in the urinary tract prevents urine excretion, leading to kidney damage.
Acute Tubular Necrosis (ATN): Intrarenal
Direct injury to kidney tubules due to ischemia or toxins.
Gentamicin (Aminoglycoside antibiotic toxicity): Intrarenal
Causes nephrotoxicity, damaging renal tubules directly.
Correct Answer is D
Explanation
(A) Sodium: Thiazide diuretics can lower sodium, but hyponatremia does not directly affect digoxin toxicity.
(B) Magnesium: Magnesium is important for cardiac function but not as directly linked to digoxin toxicity as potassium.
(C) Calcium: Thiazides can cause hypercalcemia, but calcium does not significantly affect digoxin toxicity.
(D) Potassium: Thiazide diuretics (e.g., hydrochlorothiazide) cause potassium loss, which increases the risk of digoxin toxicity. Hypokalemia sensitizes the heart to digoxin’s effects, leading to arrhythmias.
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