The nursing problem of activity Intolerance related to impaired ventricular function is made for a client with myocarditis. Which outcome in the plan of care would reflect resolution of this issue? The client will:
ambulate in the hall 3 times daily without shortness of breath.
have an increase in systolic blood pressure of 20 mmHg during activity.
decrease weight from 150 lbs. (68.2 kg.) to 148 lbs. (67.3 kg.) in five days.
verbalize the signs and symptoms of worsening heart failure by discharge.
The Correct Answer is A
A. ambulate in the hall 3 times daily without shortness of breath: This directly reflects improved tolerance to activity, the central focus of the nursing diagnosis. Being able to ambulate without dyspnea indicates better cardiac output and ventricular function, marking resolution of the identified problem.
B. have an increase in systolic blood pressure of 20 mmHg during activity: A rise in systolic pressure may occur with exertion, but it does not necessarily indicate improved activity tolerance. In fact, an exaggerated BP response could indicate the heart is working harder than it should to meet the demands, suggesting poor cardiovascular adaptation to exercise.
C. decrease weight from 150 lbs. (68.2 kg.) to 148 lbs. (67.3 kg.) in five days: A slight weight reduction may reflect decreased fluid retention, but this outcome is more relevant to volume status and fluid balance than to resolving activity intolerance specifically.
D. verbalize the signs and symptoms of worsening heart failure by discharge:
While important for client education and self-management, this outcome relates to knowledge rather than physical tolerance to activity. It does not directly measure resolution of activity intolerance.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
A. "The diaphragmatic nerve overdrives the rapid rhythm:" The phrenic (diaphragmatic) nerve controls the diaphragm for breathing and plays no role in cardiac rhythm regulation. It is unrelated to the autonomic control of heart rate or supraventricular tachycardia (SVT) management.
B. "Vagal stimulation decreases peripheral vascular resistance:" Vagal stimulation primarily affects the heart by decreasing the rate and conduction through the AV node. Its effect on vascular resistance is minimal, making this explanation inaccurate in the context of SVT treatment.
C. "The vagus nerve increases the heart rate, overdriving the rhythm:" The vagus nerve has a parasympathetic effect, slowing down the heart rate, not increasing it. Overdrive pacing is a different concept and not related to vagal maneuvers like carotid massage.
D. "The vagus nerve slows the heart rate": The carotid sinuses in the neck contain baroreceptors that are sensitive to pressure. Massaging these sinuses stimulates the vagus nerve to release acetylcholine, which in turn slows conduction through the AV node and may terminate episodes of SVT. This response demonstrates an accurate understanding of the purpose of the procedure.
Correct Answer is B
Explanation
A. Diaphoresis, hypertension: While diaphoresis may occur in response to the discomfort or anxiety caused by a rapid heart rate, hypertension is not a typical feature of atrial flutter. The rapid ventricular response can more often lead to hypotension if cardiac output is compromised.
B. Palpitations, shortness of breath: These are hallmark symptoms of rapid atrial flutter. Palpitations result from the fast, irregular atrial contractions, while shortness of breath may occur due to decreased cardiac output and poor ventricular filling during rapid rates.
C. Systolic murmur, severe anxiety: Atrial flutter is not typically associated with a systolic murmur unless there is a pre-existing valvular condition. Anxiety may be present but is a nonspecific response and not as directly linked to atrial flutter as palpitations and dyspnea.
D. Visual changes, anorexia: These symptoms are not characteristic of atrial flutter. Visual changes may occur with severe hypotension or embolic events, and anorexia may be seen in chronic illness, but neither is directly tied to the acute presentation of atrial flutter.
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