What drug would the nurse administer to treat a patient diagnosed with bronchospasm associated with chronic obstructive pulmonary disease (COPD)?
Glycopyrrolate.
Ipratropium.
Atropine.
Flavoxate.
The Correct Answer is B
Choice A rationale
Glycopyrrolate is an anticholinergic medication that reduces secretions in the mouth, throat, and airways but is not typically used for bronchospasm associated with COPD.
Choice B rationale
Ipratropium is an anticholinergic bronchodilator that is commonly used to treat bronchospasm associated with COPD. It works by relaxing the muscles around the airways to improve breathing.
Choice C rationale
Atropine is an anticholinergic medication used to treat bradycardia and reduce secretions but is not indicated for bronchospasm associated with COPD.
Choice D rationale
Flavoxate is a urinary antispasmodic used to relieve symptoms of bladder spasms but has no role in the treatment of bronchospasm associated with COPD.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A rationale
Thiazide diuretics do not block the sodium pump. They act primarily on the distal convoluted tubule in the kidneys.
Choice B rationale
Thiazide diuretics block the chloride pump in the distal convoluted tubule, preventing the reabsorption of sodium and chloride, leading to increased urine output and decreased blood pressure.
Choice C rationale
Thiazide diuretics do not block the carbonic anhydrase pump. This action is associated with carbonic anhydrase inhibitors.
Choice D rationale
Thiazide diuretics do not block the potassium pump. They can cause potassium loss as a side effect, but this is not their primary mechanism of action.
Correct Answer is D
Explanation
Choice A rationale
Cardiac glycosides are not known for increasing heart rate. Instead, they tend to decrease heart rate by enhancing vagal tone and increasing the refractory period of the atrioventricular node, which helps in controlling heart rate, especially in atrial fibrillation.
Choice B rationale
Cardiac glycosides actually increase the force of myocardial contractions. They inhibit the sodium-potassium ATPase pump, which leads to an increase in intracellular sodium and calcium levels in the myocardial cells, thus increasing the force of contraction.
Choice C rationale
Cardiac glycosides do not significantly decrease conduction velocity. They primarily affect the contractility and heart rate but their influence on conduction velocity is relatively mild.
Choice D rationale
Cardiac glycosides improve renal blood flow and increase urine output by increasing the cardiac output and reducing the activation of the renin-angiotensin-aldosterone system. This helps in relieving symptoms of heart failure.
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