What electrocardiogram (ECG) change would the healthcare professional assess for when a patient's myocardial infarction through the myocardium from the endocardium to the epicardium?

A. Increase peripheral resistance: An increase in peripheral resistance contributes to hypertension by raising the pressure in the arteries. This condition often results from vasoconstriction or structural changes in the blood vessels, leading to higher overall blood pressure.

B. Decrease in cardiac output: A decrease in cardiac output typically does not cause hypertension. In fact, low cardiac output may lead to hypotension (low blood pressure) since there is less blood being pumped into the circulatory system. This option is the exception when discussing physiological causes of hypertension.

C. Both increased cardiac output and peripheral resistance: Both increased cardiac output and peripheral resistance can lead to hypertension. An increase in either factor can elevate blood pressure, and their combined effect can significantly contribute to the development of hypertension.

D. Increased cardiac output: Increased cardiac output raises blood pressure by delivering more blood to the arteries with each heartbeat. This can occur due to various factors such as increased fluid volume, increased heart rate, or heightened contractility of the heart.

A. Vernelli's triad; Venous stasis; Hypercoagulable states; Venous intimal damage: Vernelli's triad is not a recognized term in thrombus formation. Venous stasis, hypercoagulable states, and venous intimal damage are relevant factors, but the correct terminology is Virchow's triad.

B. Vernelli's triad: Hyperlipidemia; hypercoagulable states, venous stasis: This option inaccurately references Vernelli's triad and includes hyperlipidemia, which is not one of the classic factors associated with thrombus formation. The correct factors should be named according to Virchow's triad.

C. Virchow's triad; Hyperlipidemia, Hypercoagulable states, positive D-dimer: Although Virchow's triad is correctly identified, hyperlipidemia and positive D-dimer are not part of the classic factors involved in thrombus formation. The classic factors are venous stasis, hypercoagulable states, and venous intimal damage.

D. Virchow triad; Venous stasis; Venous intimal damage; Hypercoagulable states: This option accurately identifies Virchow's triad and lists the three key factors involved in thrombus formation: venous stasis, venous intimal damage, and hypercoagulable states.