Which common adverse effect of an angiotensin II receptor antagonist will the nurse expect to assess in a patient?
Seizure
Bradycardia
Hyperkalemia
Insomnia
The Correct Answer is C
(A) Seizure: Not a typical side effect of ARBs.
(B) Bradycardia: ARBs do not significantly affect heart rate.
(C) Hyperkalemia: Angiotensin II receptor blockers (ARBs), like losartan, reduce aldosterone secretion, leading to potassium retention and hyperkalemia.
(D) Insomnia: Not a common adverse effect of ARBs.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is {"A":{"answers":"A"},"B":{"answers":"A"},"C":{"answers":"B"},"D":{"answers":"A"},"E":{"answers":"B"},"F":{"answers":"C"},"G":{"answers":"C"}}
Explanation
Prerenal |
Intrarenal |
Postrenal |
Severe Dehydration Sepsis CHF (Congestive Heart Failure) |
Acute Tubular Necrosis (ATN) Gentamicin (Aminoglycoside antibiotic toxicity) |
Benign Prostatic Hyperplasia (BPH) Renal Calculi (Kidney Stones) |
Severe Dehydration: Prerenal
Decreased blood flow to the kidneys due to low volume.
Sepsis: Prerenal
Hypotension from systemic infection reduces kidney perfusion.
CHF (Congestive Heart Failure): Prerenal
Reduced cardiac output leads to inadequate renal perfusion.
Benign Prostatic Hyperplasia (BPH): Postrenal
Urinary outflow obstruction causes back pressure on the kidneys
Renal Calculi (Kidney Stones): Postrenal
Blockage in the urinary tract prevents urine excretion, leading to kidney damage.
Acute Tubular Necrosis (ATN): Intrarenal
Direct injury to kidney tubules due to ischemia or toxins.
Gentamicin (Aminoglycoside antibiotic toxicity): Intrarenal
Causes nephrotoxicity, damaging renal tubules directly.
Correct Answer is ["B","C","D"]
Explanation
(A) Constipation: Hypocalcemia causes increased GI motility, leading to diarrhea, not constipation.
(B) Trousseau's sign: Carpal spasm when inflating a BP cuff is a classic sign of hypocalcemia.
(C) Positive Chvostek’s sign: Facial twitching when tapping the facial nerve suggests hypocalcemia.
(D) Muscle spasms: Hypocalcemia increases neuromuscular excitability, leading to spasms and cramps.
(E) Shortened QT interval: Hypocalcemia prolongs the QT interval, while hypercalcemia shortens it.
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