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Which of the following are correct about acute glomerulonephritis: (Select all that apply)

A.

Clinical manifestations: hematuria (urine is smoky, brown tinged)

B.

Clinical manifestations: oliguria

C.

Treatment: Antibiotics, corticosteroids, cytotoxic agents, anticoagulants

D.

Treatment: Prescription dose ibuprofen

E.

Clinical manifestations: Proteinuria that exceeds 3-5g/day with albumin

Answer and Explanation

Correct Answer : A,B,C

A. Clinical manifestations: hematuria (urine is smoky, brown tinged): Hematuria is a hallmark sign of acute glomerulonephritis. The smoky or cola-colored urine results from red blood cells leaking through the damaged glomeruli, often seen in post-infectious cases.

B. Clinical manifestations: oliguria: Decreased urine output is common due to impaired glomerular filtration. Oliguria reflects reduced kidney function, which contributes to fluid retention, hypertension, and accumulation of waste products.

C. Treatment: Antibiotics, corticosteroids, cytotoxic agents, anticoagulants: Depending on the cause and severity, treatment may include antibiotics for infection, corticosteroids or cytotoxic agents for inflammation, and anticoagulants if there's risk of thrombosis due to nephrotic syndrome features.

D. Treatment: Prescription dose ibuprofen: NSAIDs like ibuprofen are generally avoided in glomerulonephritis because they can reduce renal perfusion and worsen kidney injury, especially in patients already experiencing compromised kidney function.

E. Clinical manifestations: Proteinuria that exceeds 3–5g/day with albumin: This level of proteinuria is characteristic of nephrotic syndrome, not acute glomerulonephritis. While proteinuria may be present, it is typically moderate and not in the nephrotic range.


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Related Questions

Correct Answer is B

Explanation

A. Liver cirrhosis: Although chronic alcohol use can cause cirrhosis, the acute presentation with severe abdominal pain, fever, hypotension, and elevated amylase and lipase is more indicative of pancreatitis rather than cirrhosis, which typically presents with chronic symptoms.

B. Acute pancreatitis: Sudden severe abdominal pain, fever, low blood pressure, and elevated inflammatory markers like C-reactive protein along with elevated pancreatic enzymes (amylase and lipase) strongly suggest acute pancreatitis. Alcohol use is a common risk factor for this condition.

C. Hepatitis C: This usually presents with more chronic symptoms related to liver dysfunction fatigue, nausea, dark urine, and jaundice. It does not cause elevated pancreatic enzymes or acute abdominal pain.

D. Cholecystitis: While cholecystitis causes abdominal pain and fever, it does not typically cause elevated amylase and lipase levels unless there is associated pancreatitis. The vital signs and enzyme elevations point more directly to pancreatitis.

Correct Answer is A

Explanation

A. Esophageal: Esophageal variceal bleeding is the most common and serious complication of portal hypertension. Increased pressure in the portal venous system leads to the formation of varices in the esophagus, which can rupture and cause life-threatening hemorrhage.

B. Intestinal: While portal hypertension can cause changes in intestinal blood flow, it is not the most common site for clinically significant bleeding. Intestinal bleeding related to portal hypertension is much less frequent than esophageal variceal bleeding.

C. Duodenal: Duodenal bleeding is more commonly associated with peptic ulcer disease rather than portal hypertension. It is not a typical manifestation of increased portal venous pressure.

D. Rectal: Rectal bleeding may occur due to hemorrhoids or portal hypertensive colopathy, but it is less common and less life-threatening compared to esophageal varices in portal hypertension.

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