What is an ominous sign of advanced SLE?
Cognitive dysfunction from immune complex deposit in the brain
Proteinuria from early glomerulonephritis
Dysrhythmias from fibrosis of the atrioventricular node
Anemia from antibodies against blood cells
The Correct Answer is A
Choice A reason: Cognitive dysfunction in advanced systemic lupus erythematosus (SLE) results from immune complex deposition in cerebral vessels, causing neuropsychiatric symptoms like confusion or memory loss. This is an ominous sign, indicating central nervous system involvement, which is severe and potentially life-threatening due to its impact on brain function and difficulty in management.
Choice B reason: Proteinuria from early glomerulonephritis is common in SLE but indicates renal involvement, not necessarily advanced disease. It is manageable with immunosuppressants and less immediately life-threatening than cerebral involvement. While significant, it is not as ominous as cognitive dysfunction, which signals severe systemic progression.
Choice C reason: Dysrhythmias from atrioventricular node fibrosis are rare in SLE and typically occur in congenital lupus or late cardiac complications. They are not a hallmark of advanced disease compared to neuropsychiatric manifestations. This sign is less ominous, as it is less common and not indicative of widespread systemic damage.
Choice D reason: Anemia from autoantibodies against red blood cells is frequent in SLE, reflecting autoimmune activity. It is manageable with corticosteroids and not uniquely indicative of advanced disease. Cognitive dysfunction is more ominous, as it suggests severe, multisystem involvement with poor prognosis due to central nervous system damage.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Choice A reason: Increasing dietary protein and carbohydrates is not specific to SLE management. While nutrition supports overall health, SLE requires focus on photosensitivity, inflammation control, and organ protection. Recommending this risks diverting attention from critical interventions like sun protection or immunosuppression, potentially worsening SLE symptoms like rashes or flares.
Choice B reason: Nonpharmacologic pain interventions are adjunctive but not a substitute for analgesics in SLE, where pain from arthritis or organ involvement often requires medications like NSAIDs or hydroxychloroquine. Prioritizing nonpharmacologic methods alone risks inadequate pain control, reducing quality of life and misaligning with evidence-based SLE management strategies.
Choice C reason: Avoiding sunlight is critical in SLE, as ultraviolet exposure triggers photosensitive rashes and systemic flares in 70% of patients. Sun protection (sunscreen, clothing, avoiding peak hours) reduces cutaneous and systemic inflammation, preventing exacerbations. This teaching is essential for disease control, improving outcomes and patient comfort in SLE management.
Choice D reason: Genetic counseling is not routinely required for SLE family planning, as it’s not primarily hereditary, though genetic factors contribute. Counseling may be relevant for specific concerns, but it’s less critical than sun avoidance. Prioritizing this misguides teaching, potentially causing unnecessary worry and overlooking key SLE management strategies.
Correct Answer is C
Explanation
Choice A reason: Choosing red meat over poultry is incorrect, as both are high in purines, which increase uric acid levels, triggering gout attacks. Red meat, like beef, is particularly purine-rich, worsening hyperuricemia. A low-purine diet emphasizes plant-based proteins and low-purine options like eggs or dairy, indicating the client misunderstands dietary restrictions for gout management.
Choice B reason: Stating only white wine is allowed is incorrect, as all alcohol, including white wine, increases uric acid production and reduces its excretion, exacerbating gout. Beer and liquor are particularly high-risk, but no alcohol is recommended. This statement reflects a misunderstanding of the low-purine diet, which advises complete alcohol avoidance to prevent flare-ups.
Choice C reason: Avoiding liver and organ meats is correct, as these are extremely high in purines, which metabolize into uric acid, promoting gout attacks. A low-purine diet excludes organ meats, shellfish, and certain fish to reduce uric acid levels. This statement demonstrates accurate understanding of dietary restrictions essential for managing gout and preventing recurrent episodes.
Choice D reason: Not limiting fruit servings is generally true, as most fruits are low in purines. However, this statement is less specific to gout management than avoiding organ meats, which directly addresses a primary purine source. Fruits like cherries may even reduce gout risk, but this choice is less indicative of comprehensive dietary understanding.
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